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10.1016/j.bbrep.2015.11.021 http://scihub22266oqcxt.onion/10.1016/j.bbrep.2015.11.021 C5600357!5600357 !28955801     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28955801 &cmd=llinks): Failed to open stream: HTTP request failed! 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Up-regulation of Syndecan-4 contributes to TGF-?1-induced epithelial to mesenchymal transition in lung adenocarcinoma A549 cells |pdf=|usr=}}{{28955801 }} gab.com Text Up-regulation of Syndecan-4 contributes to TGF- 1-induced epithelial to mesenchymal transition in lung adenocarcinoma A549 cells JO: Biochem Biophys Rep http://www.kidney.de/pget.php?&tw=1&pmid=28955801 #FOAvip Syndecan-4 (SDC4) is a cell-surface proteoglycan associated with cell adhesion, motility, and intracellular signaling. Here, we present that SDC4 functions as a positive regulator of the transforming growth factor (TGF)-?1-induced epithelial to mesenchymal transition (EMT) via Snail in lung adenocarcinoma, A549 cells. TGF-?1 up-regulated the expression of SDC4, accompanied by the induction of EMT. Wound-healing and transwell chemotaxis assay revealed that SDC4 promoted cell migration and invasion. SDC4 knockdown recovered the E-cadherin and decreased vimentin and Snail expression in EMT-induced A549 cells. However, depletion of SDC4 resulted in little change of the Slug protein expression and mesenchymal cell morphology induced by TGF-?1. The double knockdown of SDC-4 and Slug was required for reversal of epithelial morphology; it did not occur from the SDC4 single knockdown. These findings suggest that Snail is a transcriptional factor downstream of SDC4, and SDC4 regulates TGF-?1-induced EMT by cooperating with Slug. Our data provide a novel insight into cellular mechanisms, whereby the cell-surface proteoglycan modulated TGF-?1-induced EMT in lung adenocarcinoma, A549 cells. Twit Text FOAVip Up-regulation of Syndecan-4 contributes to TGF- 1-induced epithelial to mesenchymal transition in lung adenocarcinoma A549 cells ????Biochem Biophys Rep http://www.kidney.de/pget.php?&tw=1&pmid=28955801 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28955801 #FOAvip English Wikipedia <ref>{{Cite pmid|28955801 }}</ref> Up-regulation of Syndecan-4 contributes to TGF-?1-induced epithelial to mesenchymal transition in lung adenocarcinoma A549 cells #MMPMID28955801 Toba-Ichihashi Y ; Yamaoka T ; Ohmori T ; Ohba M Biochem Biophys Rep 2016[Mar]; 5 (ä): 1-7 PMID28955801 show ga Syndecan-4 (SDC4) is a cell-surface proteoglycan associated with cell adhesion, motility, and intracellular signaling. Here, we present that SDC4 functions as a positive regulator of the transforming growth factor (TGF)-?1-induced epithelial to mesenchymal transition (EMT) via Snail in lung adenocarcinoma, A549 cells. TGF-?1 up-regulated the expression of SDC4, accompanied by the induction of EMT. Wound-healing and transwell chemotaxis assay revealed that SDC4 promoted cell migration and invasion. SDC4 knockdown recovered the E-cadherin and decreased vimentin and Snail expression in EMT-induced A549 cells. However, depletion of SDC4 resulted in little change of the Slug protein expression and mesenchymal cell morphology induced by TGF-?1. The double knockdown of SDC-4 and Slug was required for reversal of epithelial morphology; it did not occur from the SDC4 single knockdown. These findings suggest that Snail is a transcriptional factor downstream of SDC4, and SDC4 regulates TGF-?1-induced EMT by cooperating with Slug. Our data provide a novel insight into cellular mechanisms, whereby the cell-surface proteoglycan modulated TGF-?1-induced EMT in lung adenocarcinoma, A549 cells. äUp-regulation of Syndecan-4 contributes to TGF-?1-induced epithelial t(epmc).pdf DeepDyve Pubget Overpricing