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10.1038/s41598-017-11773-1 http://scihub22266oqcxt.onion/10.1038/s41598-017-11773-1 C5599509!5599509 !28912521     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28912521 &cmd=llinks): Failed to open stream: HTTP request failed! 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Advanced glycation end-product (AGE)-albumin from activated macrophage is critical in human mesenchymal stem cells survival and post-ischemic reperfusion injury |pdf=|usr=}}{{28912521 }} gab.com Text Advanced glycation end-product (AGE)-albumin from activated macrophage is critical in human mesenchymal stem cells survival and post-ischemic reperfusion injury JO: Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=28912521 #FOAvip Post-ischemic reperfusion injury (PIRI) triggers an intense inflammatory response which is essential for repair but is also implicated in pathogenesis of post-ischemic remodeling in several organs in human. Stem cell therapy has recently emerged as a promising method for treatment of PIRI in human. However, satisfactory results have not been reported due to severe loss of injected stem cells in PIRI including critical limb ischemia (CLI). For investigating the advanced glycation end-product-albumin (AGE-albumin) from activated macrophages is critical in both muscle cell and stem cell death, we evaluated the recovery of PIRI-CLI by injection of human bone marrow derived mesenchymal stem cells (hBD-MSCs) with or without soluble receptor for AGEs (sRAGE). Our results showed that activated M1 macrophages synthesize and secrete AGE-albumin, which induced the skeletal muscle cell death and injected hBD-MSCs in PIRI-CLI through RAGE increase. Combined injection of sRAGE and hBD-MSCs resulted in enhanced survival of hBD-MSCs and angiogenesis in PIRI-CLI mice. Taken together, AGE-albumin from activated macrophages is critical for both skeletal muscle cell and hBD-MSCs death in PIRI-CLI. Therefore, the inhibition of AGE-albumin from activated macrophages could be a successful therapeutic strategy for treatment of PIRI including CLI with or without stem cell therapy. Twit Text FOAVip Advanced glycation end-product (AGE)-albumin from activated macrophage is critical in human mesenchymal stem cells survival and post-ischemic reperfusion injury ????Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=28912521 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28912521 #FOAvip English Wikipedia <ref>{{Cite pmid|28912521 }}</ref> Advanced glycation end-product (AGE)-albumin from activated macrophage is critical in human mesenchymal stem cells survival and post-ischemic reperfusion injury #MMPMID28912521 Son M ; Kang WC ; Oh S ; Bayarsaikhan D ; Ahn H ; Lee J ; Park H ; Lee S ; Choi J ; Lee HS ; Yang PC ; Byun K ; Lee B Sci Rep 2017[Sep]; 7 (1 ): 11593 PMID28912521 show ga Post-ischemic reperfusion injury (PIRI) triggers an intense inflammatory response which is essential for repair but is also implicated in pathogenesis of post-ischemic remodeling in several organs in human. Stem cell therapy has recently emerged as a promising method for treatment of PIRI in human. However, satisfactory results have not been reported due to severe loss of injected stem cells in PIRI including critical limb ischemia (CLI). For investigating the advanced glycation end-product-albumin (AGE-albumin) from activated macrophages is critical in both muscle cell and stem cell death, we evaluated the recovery of PIRI-CLI by injection of human bone marrow derived mesenchymal stem cells (hBD-MSCs) with or without soluble receptor for AGEs (sRAGE). Our results showed that activated M1 macrophages synthesize and secrete AGE-albumin, which induced the skeletal muscle cell death and injected hBD-MSCs in PIRI-CLI through RAGE increase. Combined injection of sRAGE and hBD-MSCs resulted in enhanced survival of hBD-MSCs and angiogenesis in PIRI-CLI mice. Taken together, AGE-albumin from activated macrophages is critical for both skeletal muscle cell and hBD-MSCs death in PIRI-CLI. Therefore, the inhibition of AGE-albumin from activated macrophages could be a successful therapeutic strategy for treatment of PIRI including CLI with or without stem cell therapy. |*Cell Communication [MESH] |*Cell Survival [MESH] |Albumins [MESH] |Animals [MESH] |Cell Death [MESH] |Disease Models, Animal [MESH] |Glycation End Products, Advanced/*metabolism [MESH] |Humans [MESH] |Macrophage Activation/immunology [MESH] |Macrophages/immunology/*metabolism [MESH] |Male [MESH] |Mesenchymal Stem Cell Transplantation/methods [MESH] |Mesenchymal Stem Cells/*metabolism [MESH] |Mice [MESH] |Muscle Fibers, Skeletal/metabolism/pathology [MESH] |Neovascularization, Pathologic/metabolism [MESH]Advanced glycation end-product (AGE)-albumin from activated macrophage(epmc).pdf DeepDyve Pubget Overpricing