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10.4103/jomfp.JOMFP_143_17 http://scihub22266oqcxt.onion/10.4103/jomfp.JOMFP_143_17 C5596677!5596677!28932036     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Oral+Maxillofac+Pathol 2017 ; 21 (2): 260-3 Nephropedia Template TP {{tp|p=28932036|t=2017. Current concepts of pemphigus with a deep insight into its molecular aspects |pdf=|usr=}}{{28932036}} gab.com Text Current concepts of pemphigus with a deep insight into its molecular aspects JO: J Oral Maxillofac Pathol http://www.kidney.de/pget.php?&tw=1&pmid=28932036 #FOAvip Pemphigus vulgaris is an autoimmune bullous disease involving both the skin and mucosal areas, which is characterized by intraepithelial flaccid blisters and erosions. The pathogenesis of this disease is not yet completely established, but novel intuitions into its pathogenesis have recently been published. An unanswered question in its pathophysiology is the mechanism of acantholysis or loss of keratinocyte cell adhesion. Acantholysis seems to result from a communal action of autoantibodies against numerous keratinocyte self-antigens, of which desmogleins 1 and 3, desmocollins and nondesmosome components, such as the mitochondrion, might take part in the disease initiation. Lately, apoptosis was described as a possible underlying mechanism of acantholysis. Likewise, apoptolysis is assumed to be the association between suprabasal acantholytic and cell death pathways. Hence, the present review focuses on the current concepts in the pathogenesis of the pemphigus in a nutshell. Twit Text FOAVip Current concepts of pemphigus with a deep insight into its molecular aspects ????J Oral Maxillofac Pathol http://www.kidney.de/pget.php?&tw=1&pmid=28932036 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28932036 #FOAvip English Wikipedia <ref>{{Cite pmid|28932036}}</ref> Current concepts of pemphigus with a deep insight into its molecular aspects #MMPMID28932036 Madala J; Bashamalla R; Kumar MP J Oral Maxillofac Pathol 2017[May]; 21 (2): 260-3 PMID28932036show ga Pemphigus vulgaris is an autoimmune bullous disease involving both the skin and mucosal areas, which is characterized by intraepithelial flaccid blisters and erosions. The pathogenesis of this disease is not yet completely established, but novel intuitions into its pathogenesis have recently been published. An unanswered question in its pathophysiology is the mechanism of acantholysis or loss of keratinocyte cell adhesion. Acantholysis seems to result from a communal action of autoantibodies against numerous keratinocyte self-antigens, of which desmogleins 1 and 3, desmocollins and nondesmosome components, such as the mitochondrion, might take part in the disease initiation. Lately, apoptosis was described as a possible underlying mechanism of acantholysis. Likewise, apoptolysis is assumed to be the association between suprabasal acantholytic and cell death pathways. Hence, the present review focuses on the current concepts in the pathogenesis of the pemphigus in a nutshell. äCurrent concepts of pemphigus with a deep insight into its molecular a(epmc).pdf DeepDyve Pubget Overpricing