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10.1586/eci.12.58 http://scihub22266oqcxt.onion/10.1586/eci.12.58 C5595352!5595352!23078063     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Expert+Rev+Clin+Immunol 2012 ; 8 (7): 663-72 Nephropedia Template TP {{tp|p=23078063|t=2012. Chronic rejection: a significant role for Th17-mediated autoimmune responses to self-antigens |pdf=|usr=}}{{23078063}} gab.com Text Chronic rejection: a significant role for Th17-mediated autoimmune responses to self-antigens JO: Expert Rev Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=23078063 #FOAvip Despite progress in the field of organ transplantation for improvement in graft survival and function, long-term graft function is still limited by the development of chronic allograft rejection. Various immune-mediated and nonimmune-mediated processes have been postulated in the pathogenesis of chronic rejection. In this review, the authors discuss the important role of alloimmune responses to donor-specific antigens and autoimmune responses to tissue restricted self-antigens in the immunopathogenesis of chronic rejection following solid organ transplantation. In particular, the authors discuss the role of induction of Th17-type autoimmune responses and the crosstalk between autoimmune and alloimmune responses. These self-perpetuate each other leading to activation of profibrotic and proinflammatory cascades that ultimately result in the development of chronic rejection. Twit Text FOAVip Chronic rejection: a significant role for Th17-mediated autoimmune responses to self-antigens ????Expert Rev Clin Immunol http://www.kidney.de/pget.php?&tw=1&pmid=23078063 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=23078063 #FOAvip English Wikipedia <ref>{{Cite pmid|23078063}}</ref> Chronic rejection: a significant role for Th17-mediated autoimmune responses to self-antigens #MMPMID23078063 Subramanian V; Mohanakumar T Expert Rev Clin Immunol 2012[Sep]; 8 (7): 663-72 PMID23078063show ga Despite progress in the field of organ transplantation for improvement in graft survival and function, long-term graft function is still limited by the development of chronic allograft rejection. Various immune-mediated and nonimmune-mediated processes have been postulated in the pathogenesis of chronic rejection. In this review, the authors discuss the important role of alloimmune responses to donor-specific antigens and autoimmune responses to tissue restricted self-antigens in the immunopathogenesis of chronic rejection following solid organ transplantation. In particular, the authors discuss the role of induction of Th17-type autoimmune responses and the crosstalk between autoimmune and alloimmune responses. These self-perpetuate each other leading to activation of profibrotic and proinflammatory cascades that ultimately result in the development of chronic rejection. äChronic rejection: a significant role for Th17-mediated autoimmune res(epmc).pdf DeepDyve Pubget Overpricing