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10.3892/ol.2017.6729 http://scihub22266oqcxt.onion/10.3892/ol.2017.6729 C5592856!5592856!28943961     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Oncol+Lett 2017 ; 14 (4): 4613-8 Nephropedia Template TP {{tp|p=28943961|t=2017. The roles of ARHGAP10 in the proliferation, migration and invasion of lung cancer cells |pdf=|usr=}}{{28943961}} gab.com Text The roles of ARHGAP10 in the proliferation, migration and invasion of lung cancer cells JO: Oncol Lett http://www.kidney.de/pget.php?&tw=1&pmid=28943961 #FOAvip Lung cancer is a leading cause of cancer-related mortalities worldwide. In the present study, a comparison of To determine the roles of ARHGAP10 in the proliferation, migration and invasion of lung cancer cells expression levels between normal lung tissues and lung cancer tissues were compared using immunoblotting, and CCK-8 and Transwell assays. Lung cancer tissues had a decreased ARHGAP10 mRNA expression level compared to the adjacent normal tissues. The ectopic expression of ARHGAP10 significantly suppressed the migration, invasion and proliferation of lung cancer cells. Gene set enrichment analysis revealed that metastasis and Wnt signaling pathways were negatively correlated with ARHGAP10 expression. Immunoblotting analysis revealed that ARHGAP10 overexpression inhibited metastasis [matrix metalloproteinase (MMP)-2, MMP-9 and VEGF] and the expression of Wnt pathway-related proteins (?-catenin and c-Myc). Moreover, the stimulation effects of lithium chloride, a GSK3? inhibitor, on the accumulation of ?-catenin were notably suppressed by ARHGAP10 overexpression. Collectively, ARHGAP10 acts to suppress tumor within lung cancer by affecting metastasis and Wnt signaling pathways. The results therefore suggest that ARHGAP10 is a potentially attractive target for the treatment of lung cancer. Twit Text FOAVip The roles of ARHGAP10 in the proliferation, migration and invasion of lung cancer cells ????Oncol Lett http://www.kidney.de/pget.php?&tw=1&pmid=28943961 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28943961 #FOAvip English Wikipedia <ref>{{Cite pmid|28943961}}</ref> The roles of ARHGAP10 in the proliferation, migration and invasion of lung cancer cells #MMPMID28943961 Teng JP; Yang ZY; Zhu YM; Ni D; Zhu ZJ; Li XQ Oncol Lett 2017[Oct]; 14 (4): 4613-8 PMID28943961show ga Lung cancer is a leading cause of cancer-related mortalities worldwide. In the present study, a comparison of To determine the roles of ARHGAP10 in the proliferation, migration and invasion of lung cancer cells expression levels between normal lung tissues and lung cancer tissues were compared using immunoblotting, and CCK-8 and Transwell assays. Lung cancer tissues had a decreased ARHGAP10 mRNA expression level compared to the adjacent normal tissues. The ectopic expression of ARHGAP10 significantly suppressed the migration, invasion and proliferation of lung cancer cells. Gene set enrichment analysis revealed that metastasis and Wnt signaling pathways were negatively correlated with ARHGAP10 expression. Immunoblotting analysis revealed that ARHGAP10 overexpression inhibited metastasis [matrix metalloproteinase (MMP)-2, MMP-9 and VEGF] and the expression of Wnt pathway-related proteins (?-catenin and c-Myc). Moreover, the stimulation effects of lithium chloride, a GSK3? inhibitor, on the accumulation of ?-catenin were notably suppressed by ARHGAP10 overexpression. Collectively, ARHGAP10 acts to suppress tumor within lung cancer by affecting metastasis and Wnt signaling pathways. The results therefore suggest that ARHGAP10 is a potentially attractive target for the treatment of lung cancer. äThe roles of ARHGAP10 in the proliferation, migration and invasion of (epmc).pdf DeepDyve Pubget Overpricing