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10.3892/ol.2017.6600 http://scihub22266oqcxt.onion/10.3892/ol.2017.6600 C5588050!5588050 !28927149     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28927149 &cmd=llinks): Failed to open stream: HTTP request failed! 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MicroRNA-552 promotes tumor cell proliferation and migration by directly targeting DACH1 via the Wnt/?-catenin signaling pathway in colorectal cancer |pdf=|usr=}}{{28927149 }} gab.com Text MicroRNA-552 promotes tumor cell proliferation and migration by directly targeting DACH1 via the Wnt/ -catenin signaling pathway in colorectal cancer JO: Oncol Lett http://www.kidney.de/pget.php?&tw=1&pmid=28927149 #FOAvip The purpose of the present study was to analyze the crucial role of microRNAs (miRNAs/miRs) involved in the proliferation and migration of colorectal cancer (CRC) and to investigate their underlying mechanisms. The present study discusses the expression and function of miR-552 in CRC. The expression level of miR-552 in CRC cells and tissues was observed, and it was suggested that the high expression of miR-552 accelerated the proliferation and migration of CRC cells in vitro. Notably, a result of the present study was that the cell fate determination factor Dachshund family transcription factor 1 (DACH1) was identified as a direct target of miR-552. Suppressing miR-552 expression in CRC cells increased endogenous DACH1 mRNA and protein levels, which was negatively correlated with miR-552. DACH1 performs an important role in the development of a number of neoplasms, and has the ability to regulate the Wnt/?-catenin signaling pathway as a novel predictive and diagnostic biomarker. Accordingly, it was concluded that miR-552 exerted a tumor-promoting role in CRC development by targeting DACH1, which may contribute to the increase in the rates of CRC proliferation and migration. miR-552 may serve as a potential diagnostic and prognostic biomarker for CRC. Twit Text FOAVip MicroRNA-552 promotes tumor cell proliferation and migration by directly targeting DACH1 via the Wnt/ -catenin signaling pathway in colorectal cancer ????Oncol Lett http://www.kidney.de/pget.php?&tw=1&pmid=28927149 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28927149 #FOAvip English Wikipedia <ref>{{Cite pmid|28927149 }}</ref> MicroRNA-552 promotes tumor cell proliferation and migration by directly targeting DACH1 via the Wnt/?-catenin signaling pathway in colorectal cancer #MMPMID28927149 Cao J ; Yan XR ; Liu T ; Han XB ; Yu JJ ; Liu SH ; Wang LB Oncol Lett 2017[Sep]; 14 (3 ): 3795-3802 PMID28927149 show ga The purpose of the present study was to analyze the crucial role of microRNAs (miRNAs/miRs) involved in the proliferation and migration of colorectal cancer (CRC) and to investigate their underlying mechanisms. The present study discusses the expression and function of miR-552 in CRC. The expression level of miR-552 in CRC cells and tissues was observed, and it was suggested that the high expression of miR-552 accelerated the proliferation and migration of CRC cells in vitro. Notably, a result of the present study was that the cell fate determination factor Dachshund family transcription factor 1 (DACH1) was identified as a direct target of miR-552. Suppressing miR-552 expression in CRC cells increased endogenous DACH1 mRNA and protein levels, which was negatively correlated with miR-552. DACH1 performs an important role in the development of a number of neoplasms, and has the ability to regulate the Wnt/?-catenin signaling pathway as a novel predictive and diagnostic biomarker. Accordingly, it was concluded that miR-552 exerted a tumor-promoting role in CRC development by targeting DACH1, which may contribute to the increase in the rates of CRC proliferation and migration. miR-552 may serve as a potential diagnostic and prognostic biomarker for CRC. äMicroRNA-552 promotes tumor cell proliferation and migration by direct(epmc).pdf DeepDyve Pubget Overpricing