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10.1155/2017/2683120 http://scihub22266oqcxt.onion/10.1155/2017/2683120 C5587962!5587962!28912982     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Case+Rep+Endocrinol 2017 ; 2017 (ä): ä Nephropedia Template TP {{tp|p=28912982|t=2017. Severe Thyrotoxicosis Secondary to Povidone-Iodine from Peritoneal Dialysis |pdf=|usr=}}{{28912982}} gab.com Text Severe Thyrotoxicosis Secondary to Povidone-Iodine from Peritoneal Dialysis JO: Case Rep Endocrinol http://www.kidney.de/pget.php?&tw=1&pmid=28912982 #FOAvip A 73-year-old male on home peritoneal dialysis (PD) with recent diagnosis of atrial fibrillation presented with fatigue and dyspnea. Hyperthyroidism was diagnosed with TSH < 0.01?mIU/L and FT4 > 100?pmol/L. He had no personal or family history of thyroid disease. There had been no exposures to CT contrast, amiodarone, or iodine. Technetium thyroid scan showed diffusely decreased uptake. He was discharged with a presumptive diagnosis of thyroiditis. Three weeks later, he had deteriorated clinically. Possible iodine sources were again reviewed, and it was determined that povidone-iodine solution was used with each PD cycle. Methimazole 25?mg daily was initiated; however, he had difficulty tolerating the medication and continued to clinically deteriorate. He was readmitted to hospital where methimazole was restarted at 20?mg bid with high dose prednisone 25?mg and daily plasma exchange (PLEX) therapy. Biochemical improvement was observed with FT4 dropping to 48.5?pmol/L by day 10, but FT4 rebounded to 67.8?pmol/L after PLEX was discontinued. PLEX was restarted and thyroidectomy was performed. Pathology revealed nodular hyperplasia with no evidence of thyroiditis. Preoperative plasma iodine levels were greater than 5 times the upper limit of normal range. We hypothesize that the patient had underlying autonomous thyroid hormone production exacerbated by exogenous iodine exposure from a previously unreported PD-related source. Twit Text FOAVip Severe Thyrotoxicosis Secondary to Povidone-Iodine from Peritoneal Dialysis ????Case Rep Endocrinol http://www.kidney.de/pget.php?&tw=1&pmid=28912982 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28912982 #FOAvip English Wikipedia <ref>{{Cite pmid|28912982}}</ref> Severe Thyrotoxicosis Secondary to Povidone-Iodine from Peritoneal Dialysis #MMPMID28912982 Lithgow K; Symonds C Case Rep Endocrinol 2017[]; 2017 (ä): ä PMID28912982show ga A 73-year-old male on home peritoneal dialysis (PD) with recent diagnosis of atrial fibrillation presented with fatigue and dyspnea. Hyperthyroidism was diagnosed with TSH < 0.01?mIU/L and FT4 > 100?pmol/L. He had no personal or family history of thyroid disease. There had been no exposures to CT contrast, amiodarone, or iodine. Technetium thyroid scan showed diffusely decreased uptake. He was discharged with a presumptive diagnosis of thyroiditis. Three weeks later, he had deteriorated clinically. Possible iodine sources were again reviewed, and it was determined that povidone-iodine solution was used with each PD cycle. Methimazole 25?mg daily was initiated; however, he had difficulty tolerating the medication and continued to clinically deteriorate. He was readmitted to hospital where methimazole was restarted at 20?mg bid with high dose prednisone 25?mg and daily plasma exchange (PLEX) therapy. Biochemical improvement was observed with FT4 dropping to 48.5?pmol/L by day 10, but FT4 rebounded to 67.8?pmol/L after PLEX was discontinued. PLEX was restarted and thyroidectomy was performed. Pathology revealed nodular hyperplasia with no evidence of thyroiditis. Preoperative plasma iodine levels were greater than 5 times the upper limit of normal range. We hypothesize that the patient had underlying autonomous thyroid hormone production exacerbated by exogenous iodine exposure from a previously unreported PD-related source. äSevere Thyrotoxicosis Secondary to Povidone-Iodine from Peritoneal Dia(epmc).pdf DeepDyve Pubget Overpricing