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10.1002/2211-5463.12279 http://scihub22266oqcxt.onion/10.1002/2211-5463.12279 C5586351!5586351!28904868     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       FEBS+Open+Bio 2017 ; 7 (9): 1402-9 Nephropedia Template TP {{tp|p=28904868|t=2017. High?density lipoprotein protects cardiomyocytes from oxidative stress via the PI3K/mTOR signaling pathway |pdf=|usr=}}{{28904868}} gab.com Text High density lipoprotein protects cardiomyocytes from oxidative stress via the PI3K/mTOR signaling pathway JO: FEBS Open Bio http://www.kidney.de/pget.php?&tw=1&pmid=28904868 #FOAvip Low levels of plasma high?density lipoprotein (HDL) cholesterol are associated with an increased risk of heart failure, regardless of the presence or absence of coronary artery disease. However, the direct effects of HDL on failing myocardium have not been fully elucidated. We found that HDL treatment resulted in improved cell viability in H9c2 cardiomyocytes under oxidative stress. This cardioprotective effect of HDL was regulated via the phosphatidylinositol 3?kinase (PI3K)/mammalian target of rapamycin (mTOR) pathway. mTOR signaling promotes cell survival through the inactivation of the BCL2?associated agonist of cell death via phosphorylation of ribosomal protein S6 kinase. Modulation of cardiac PI3K/mTOR signaling by HDL could represent a novel therapeutic strategy for heart failure. Twit Text FOAVip High density lipoprotein protects cardiomyocytes from oxidative stress via the PI3K/mTOR signaling pathway ????FEBS Open Bio http://www.kidney.de/pget.php?&tw=1&pmid=28904868 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28904868 #FOAvip English Wikipedia <ref>{{Cite pmid|28904868}}</ref> High?density lipoprotein protects cardiomyocytes from oxidative stress via the PI3K/mTOR signaling pathway #MMPMID28904868 Nagao M; Toh R; Irino Y; Nakajima H; Oshita T; Tsuda S; Hara T; Shinohara M; Ishida T; Hirata K FEBS Open Bio 2017[Sep]; 7 (9): 1402-9 PMID28904868show ga Low levels of plasma high?density lipoprotein (HDL) cholesterol are associated with an increased risk of heart failure, regardless of the presence or absence of coronary artery disease. However, the direct effects of HDL on failing myocardium have not been fully elucidated. We found that HDL treatment resulted in improved cell viability in H9c2 cardiomyocytes under oxidative stress. This cardioprotective effect of HDL was regulated via the phosphatidylinositol 3?kinase (PI3K)/mammalian target of rapamycin (mTOR) pathway. mTOR signaling promotes cell survival through the inactivation of the BCL2?associated agonist of cell death via phosphorylation of ribosomal protein S6 kinase. Modulation of cardiac PI3K/mTOR signaling by HDL could represent a novel therapeutic strategy for heart failure. äHigh?density lipoprotein protects cardiomyocytes from oxidative stress(epmc).pdf DeepDyve Pubget Overpricing