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10.18632/oncotarget.16926 http://scihub22266oqcxt.onion/10.18632/oncotarget.16926 C5584185!5584185!28881592     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Oncotarget 2017 ; 8 (31): 50665-72 Nephropedia Template TP {{tp|p=28881592|t=2017. Associations between LMO1 gene polymorphisms and Wilms tumor susceptibility |pdf=|usr=}}{{28881592}} gab.com Text Associations between LMO1 gene polymorphisms and Wilms tumor susceptibility JO: Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=28881592 #FOAvip Wilms? tumor is the most common childhood renal malignancy. A genome-wide association study identified LIM domain only 1 (LMO1) as having oncogenic potential. We examined the associations between LMO1 gene polymorphisms and susceptibility to Wilms? tumor. In this hospital-based, case-control study, we recruited 145 children with Wilms? tumor and 531 cancer-free children. Four polymorphisms (rs110419 A>G, rs4758051 G>A, rs10840002 A>G and rs204938 A>G) were genotyped using Taqman methodology. Odds ratios (ORs) and 95% confidence intervals (CIs) were calculated to measure the associations between selected polymorphisms and Wilms? tumor susceptibility. Only rs110419 AG was found to be protective against Wilms? tumor (adjusted OR = 0.62, 95% CI = 0.41?0.94, P = 0.024) when compared to rs110419 AA. Wilms? tumor risk was markedly greater in children with 1?4 risk genotypes (nucleotide alterations) than in those with no risk genotypes (adjusted OR = 1.84, 95% CI = 1.25?2.69, P = 0.002). In a stratified analysis, the protective effect of rs110419 AG/GG was predominant in males. The association of 1?4 risk genotypes with Wilms? tumor risk was limited to subgroups of children who were >18 months old, female, and in clinical stages III+IV. Thus, LMO1 gene polymorphisms may contribute to Wilms? tumor risk, but this conclusion should be validated in other populations and larger studies. Twit Text FOAVip Associations between LMO1 gene polymorphisms and Wilms tumor susceptibility ????Oncotarget http://www.kidney.de/pget.php?&tw=1&pmid=28881592 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28881592 #FOAvip English Wikipedia <ref>{{Cite pmid|28881592}}</ref> Associations between LMO1 gene polymorphisms and Wilms tumor susceptibility #MMPMID28881592 Liu GC; Zhuo ZJ; Zhu SB; Zhu J; Jia W; Zhao Z; Hu JH; He J; Wang FH; Fu W Oncotarget 2017[Aug]; 8 (31): 50665-72 PMID28881592show ga Wilms? tumor is the most common childhood renal malignancy. A genome-wide association study identified LIM domain only 1 (LMO1) as having oncogenic potential. We examined the associations between LMO1 gene polymorphisms and susceptibility to Wilms? tumor. In this hospital-based, case-control study, we recruited 145 children with Wilms? tumor and 531 cancer-free children. Four polymorphisms (rs110419 A>G, rs4758051 G>A, rs10840002 A>G and rs204938 A>G) were genotyped using Taqman methodology. Odds ratios (ORs) and 95% confidence intervals (CIs) were calculated to measure the associations between selected polymorphisms and Wilms? tumor susceptibility. Only rs110419 AG was found to be protective against Wilms? tumor (adjusted OR = 0.62, 95% CI = 0.41?0.94, P = 0.024) when compared to rs110419 AA. Wilms? tumor risk was markedly greater in children with 1?4 risk genotypes (nucleotide alterations) than in those with no risk genotypes (adjusted OR = 1.84, 95% CI = 1.25?2.69, P = 0.002). In a stratified analysis, the protective effect of rs110419 AG/GG was predominant in males. The association of 1?4 risk genotypes with Wilms? tumor risk was limited to subgroups of children who were >18 months old, female, and in clinical stages III+IV. Thus, LMO1 gene polymorphisms may contribute to Wilms? tumor risk, but this conclusion should be validated in other populations and larger studies. äAssociations between LMO1 gene polymorphisms and Wilms tumor suscepti(epmc).pdf DeepDyve Pubget Overpricing