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10.1016/j.jacbts.2017.03.014 http://scihub22266oqcxt.onion/10.1016/j.jacbts.2017.03.014 C5582195!5582195 !28920098     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28920098 &cmd=llinks): Failed to open stream: HTTP request failed! 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The IL-1RI Co-Receptor TILRR (FREM1 Isoform 2) Controls Aberrant Inflammatory Responses and Development of Vascular Disease |pdf=|usr=}}{{28920098 }} gab.com Text The IL-1RI Co-Receptor TILRR (FREM1 Isoform 2) Controls Aberrant Inflammatory Responses and Development of Vascular Disease JO: JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=28920098 #FOAvip Expression of the interleukin-1 receptor type I (IL-1RI) co-receptor Toll-like and interleukin-1 receptor regulator (TILRR) is significantly increased in blood monocytes following myocardial infarction and in the atherosclerotic plaque, whereas levels in healthy tissue are low. TILRR association with IL-1RI at these sites causes aberrant activation of inflammatory genes, which underlie progression of cardiovascular disease. The authors show that genetic deletion of TILRR or antibody blocking of TILRR function reduces development of atherosclerotic plaques. Lesions exhibit decreased levels of monocytes, with increases in collagen and smooth muscle cells, characteristic features of stable plaques. The results suggest that TILRR may constitute a rational target for site- and signal-specific inhibition of vascular disease. Twit Text FOAVip The IL-1RI Co-Receptor TILRR (FREM1 Isoform 2) Controls Aberrant Inflammatory Responses and Development of Vascular Disease ????JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=28920098 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28920098 #FOAvip English Wikipedia <ref>{{Cite pmid|28920098 }}</ref> The IL-1RI Co-Receptor TILRR (FREM1 Isoform 2) Controls Aberrant Inflammatory Responses and Development of Vascular Disease #MMPMID28920098 Smith SA ; Samokhin AO ; Alfadi M ; Murphy EC ; Rhodes D ; Holcombe WML ; Kiss-Toth E ; Storey RF ; Yee SP ; Francis SE ; Qwarnstrom EE JACC Basic Transl Sci 2017[Aug]; 2 (4 ): 398-414 PMID28920098 show ga Expression of the interleukin-1 receptor type I (IL-1RI) co-receptor Toll-like and interleukin-1 receptor regulator (TILRR) is significantly increased in blood monocytes following myocardial infarction and in the atherosclerotic plaque, whereas levels in healthy tissue are low. TILRR association with IL-1RI at these sites causes aberrant activation of inflammatory genes, which underlie progression of cardiovascular disease. The authors show that genetic deletion of TILRR or antibody blocking of TILRR function reduces development of atherosclerotic plaques. Lesions exhibit decreased levels of monocytes, with increases in collagen and smooth muscle cells, characteristic features of stable plaques. The results suggest that TILRR may constitute a rational target for site- and signal-specific inhibition of vascular disease. äThe IL-1RI Co-Receptor TILRR (FREM1 Isoform 2) Controls Aberrant Infla(epmc).pdf DeepDyve Pubget Overpricing