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10.1111/apha.12764 http://scihub22266oqcxt.onion/10.1111/apha.12764 C5575831!5575831!27474473     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Acta+Physiol+(Oxf) 2017 ; 219 (3): 554-72 Nephropedia Template TP {{tp|p=27474473|t=2017. Biomarkers in acute kidney injury ? pathophysiological basis and clinical performance |pdf=|usr=}}{{27474473}} gab.com Text Biomarkers in acute kidney injury pathophysiological basis and clinical performance JO: Acta Physiol (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=27474473 #FOAvip Various biomarkers of acute kidney injury (AKI) have been discovered and characterized in the recent past. These molecules can be detected in urine or blood and signify structural damage to the kidney. Clinically, they are proposed as adjunct diagnostics to serum creatinine and urinary output to improve the early detection, differential diagnosis and prognostic assessment of AKI. The most obvious requirements for a biomarker include its reflection of the underlying pathophysiology of the disease. Hence, a biomarker of AKI should derive from the injured kidney and reflect a molecular process intimately connected with tissue injury. Here, we provide an overview of the basic pathophysiology, the cellular sources and the clinical performance of the most important currently proposed biomarkers of AKI: neutrophil gelatinase-associated lipocalin (NGAL), kidney injury molecule-1 (KIM-1), liver-type fatty acid-binding protein (L-FABP), interleukin-18 (IL-18), insulin-like growth factor-binding protein 7 (IGFBP7), tissue inhibitor of metalloproteinase 2 (TIMP-2) and calprotectin (S100A8/9). We also acknowledge each biomarker?s advantages and disadvantages as well as important knowledge gaps and perspectives for future studies. Twit Text FOAVip Biomarkers in acute kidney injury pathophysiological basis and clinical performance ????Acta Physiol (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=27474473 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27474473 #FOAvip English Wikipedia <ref>{{Cite pmid|27474473}}</ref> Biomarkers in acute kidney injury ? pathophysiological basis and clinical performance #MMPMID27474473 Schrezenmeier E; Barasch J; Budde K; Westhoff T; Schmidt-Ott K Acta Physiol (Oxf) 2017[Mar]; 219 (3): 554-72 PMID27474473show ga Various biomarkers of acute kidney injury (AKI) have been discovered and characterized in the recent past. These molecules can be detected in urine or blood and signify structural damage to the kidney. Clinically, they are proposed as adjunct diagnostics to serum creatinine and urinary output to improve the early detection, differential diagnosis and prognostic assessment of AKI. The most obvious requirements for a biomarker include its reflection of the underlying pathophysiology of the disease. Hence, a biomarker of AKI should derive from the injured kidney and reflect a molecular process intimately connected with tissue injury. Here, we provide an overview of the basic pathophysiology, the cellular sources and the clinical performance of the most important currently proposed biomarkers of AKI: neutrophil gelatinase-associated lipocalin (NGAL), kidney injury molecule-1 (KIM-1), liver-type fatty acid-binding protein (L-FABP), interleukin-18 (IL-18), insulin-like growth factor-binding protein 7 (IGFBP7), tissue inhibitor of metalloproteinase 2 (TIMP-2) and calprotectin (S100A8/9). We also acknowledge each biomarker?s advantages and disadvantages as well as important knowledge gaps and perspectives for future studies. äBiomarkers in acute kidney injury ? pathophysiological basis and clini(epmc).pdf DeepDyve Pubget Overpricing