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10.1097/MOG.0000000000000363 http://scihub22266oqcxt.onion/10.1097/MOG.0000000000000363 C5572460!5572460!28402994     free     free     free       Curr+Opin+Gastroenterol 2017 ; 33 (4): 239-45 Nephropedia Template TP {{tp|p=28402994|t=2017. Intestinal fibrosis: Ready to be Reversed |pdf=|usr=}}{{28402994}} gab.com Text Intestinal fibrosis: Ready to be Reversed JO: Curr Opin Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=28402994 #FOAvip Purpose of the review: Intestinal fibrosis is a common complication of several enteropathies with inflammatory bowel disease being the major cause. Intestinal fibrosis affects both ulcerative colitis and Crohn?s disease, and no specific anti-fibrotic therapy exists. This review highlights recent developments in this area. Recent findings: The pathophysiology of intestinal stricture formation includes inflammation dependent and independent mechanisms. A better understanding of the mechanisms of intestinal fibrogenesis and the availability of compounds for other, non intestinal fibrotic diseases bring clincial trials in stricturing Crohn?s disease within reach. Summary: Improved understanding of its mechanisms and ongoing development of clincial trial endpoints for intestinal fibrosis will allow the testing of novel anti-fibrotic compounds in inflammatory bowel disease. Twit Text FOAVip Intestinal fibrosis: Ready to be Reversed ????Curr Opin Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=28402994 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28402994 #FOAvip English Wikipedia <ref>{{Cite pmid|28402994}}</ref> Intestinal fibrosis: Ready to be Reversed #MMPMID28402994 Latella G; Rieder F Curr Opin Gastroenterol 2017[Jul]; 33 (4): 239-45 PMID28402994show ga Purpose of the review: Intestinal fibrosis is a common complication of several enteropathies with inflammatory bowel disease being the major cause. Intestinal fibrosis affects both ulcerative colitis and Crohn?s disease, and no specific anti-fibrotic therapy exists. This review highlights recent developments in this area. Recent findings: The pathophysiology of intestinal stricture formation includes inflammation dependent and independent mechanisms. A better understanding of the mechanisms of intestinal fibrogenesis and the availability of compounds for other, non intestinal fibrotic diseases bring clincial trials in stricturing Crohn?s disease within reach. Summary: Improved understanding of its mechanisms and ongoing development of clincial trial endpoints for intestinal fibrosis will allow the testing of novel anti-fibrotic compounds in inflammatory bowel disease. äIntestinal fibrosis: Ready to be Reversed (epmc).pdf DeepDyve Pubget Overpricing