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10.1080/21688370.2017.1356900 http://scihub22266oqcxt.onion/10.1080/21688370.2017.1356900 C5571778!5571778!28783415     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Tissue+Barriers 2017 ; 5 (3): ä Nephropedia Template TP {{tp|p=28783415|t=2017. Cadherin-mediated cell-cell interactions in normal and cancer cells |pdf=|usr=}}{{28783415}} gab.com Text Cadherin-mediated cell-cell interactions in normal and cancer cells JO: Tissue Barriers http://www.kidney.de/pget.php?&tw=1&pmid=28783415 #FOAvip Adherens junctions (AJs) are molecular complexes that mediate cell-cell adhesive interactions and play pivotal roles in maintenance of tissue organization in adult organisms and at various stages of development. AJs consist of cadherin adhesion receptors, providing homophilic ligation with cadherins on adjacent cells, and members of the catenin protein family: p120, ?- and ?-catenin. ?-catenin's linkage with the actin cytoskeleton defines the linear or punctate organization of AJs in different cell types. Myosin II-dependent tension drives vinculin recruitment by ?-catenin and stabilizes the linkage of the cadherin/catenin complex to F-actin. Neoplastic transformation leads to prominent changes in the organization, regulation and stability of AJs. Epithelial-mesenchymal transition (EMT) whereby epithelial cells lose stable cell-cell adhesion, and reorganize their cytoskeleton to acquire migratory activity, plays the central role in cancer cell invasion and metastasis. Recent data demonstrated that a partial EMT resulting in a hybrid epithelial/mesenchymal phenotype with retention of E-cadherin is essential for cancer cell dissemination. E-cadherin and E-cadherin-based AJs are required for collective invasion and migration, survival in circulation, and metastatic outgrowth. Twit Text FOAVip Cadherin-mediated cell-cell interactions in normal and cancer cells ????Tissue Barriers http://www.kidney.de/pget.php?&tw=1&pmid=28783415 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28783415 #FOAvip English Wikipedia <ref>{{Cite pmid|28783415}}</ref> Cadherin-mediated cell-cell interactions in normal and cancer cells #MMPMID28783415 Gloushankova NA; Rubtsova SN; Zhitnyak IY Tissue Barriers 2017[]; 5 (3): ä PMID28783415show ga Adherens junctions (AJs) are molecular complexes that mediate cell-cell adhesive interactions and play pivotal roles in maintenance of tissue organization in adult organisms and at various stages of development. AJs consist of cadherin adhesion receptors, providing homophilic ligation with cadherins on adjacent cells, and members of the catenin protein family: p120, ?- and ?-catenin. ?-catenin's linkage with the actin cytoskeleton defines the linear or punctate organization of AJs in different cell types. Myosin II-dependent tension drives vinculin recruitment by ?-catenin and stabilizes the linkage of the cadherin/catenin complex to F-actin. Neoplastic transformation leads to prominent changes in the organization, regulation and stability of AJs. Epithelial-mesenchymal transition (EMT) whereby epithelial cells lose stable cell-cell adhesion, and reorganize their cytoskeleton to acquire migratory activity, plays the central role in cancer cell invasion and metastasis. Recent data demonstrated that a partial EMT resulting in a hybrid epithelial/mesenchymal phenotype with retention of E-cadherin is essential for cancer cell dissemination. E-cadherin and E-cadherin-based AJs are required for collective invasion and migration, survival in circulation, and metastatic outgrowth. äCadherin-mediated cell-cell interactions in normal and cancer cells (epmc).pdf DeepDyve Pubget Overpricing