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10.3389/fonc.2017.00174 http://scihub22266oqcxt.onion/10.3389/fonc.2017.00174 C5563315!5563315!28868254     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Oncol 2017 ; 7 (ä): ä Nephropedia Template TP {{tp|p=28868254|t=2017. Mitochondria-Associated Membranes As Networking Platforms and Regulators of Cancer Cell Fate |pdf=|usr=}}{{28868254}} gab.com Text Mitochondria-Associated Membranes As Networking Platforms and Regulators of Cancer Cell Fate JO: Front Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28868254 #FOAvip The tight cross talk between two essential organelles of the cell, the endoplasmic reticulum (ER) and mitochondria, is spatially and functionally regulated by specific microdomains known as the mitochondria-associated membranes (MAMs). MAMs are hot spots of Ca2+ transfer between the ER and mitochondria, and emerging data indicate their vital role in the regulation of fundamental physiological processes, chief among them mitochondria bioenergetics, proteostasis, cell death, and autophagy. Moreover, and perhaps not surprisingly, it has become clear that signaling events regulated at the ER?mitochondria intersection regulate key processes in oncogenesis and in the response of cancer cells to therapeutics. ER?mitochondria appositions have been shown to dynamically recruit oncogenes and tumor suppressors, modulating their activity and protein complex formation, adapt the bioenergetic demand of cancer cells and to regulate cell death pathways and redox signaling in cancer cells. In this review, we discuss some emerging players of the ER?mitochondria contact sites in mammalian cells, the key processes they regulate and recent evidence highlighting the role of MAMs in shaping cell-autonomous and non-autonomous signals that regulate cancer growth. Twit Text FOAVip Mitochondria-Associated Membranes As Networking Platforms and Regulators of Cancer Cell Fate ????Front Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28868254 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28868254 #FOAvip English Wikipedia <ref>{{Cite pmid|28868254}}</ref> Mitochondria-Associated Membranes As Networking Platforms and Regulators of Cancer Cell Fate #MMPMID28868254 Sassano ML; van Vliet AR; Agostinis P Front Oncol 2017[]; 7 (ä): ä PMID28868254show ga The tight cross talk between two essential organelles of the cell, the endoplasmic reticulum (ER) and mitochondria, is spatially and functionally regulated by specific microdomains known as the mitochondria-associated membranes (MAMs). MAMs are hot spots of Ca2+ transfer between the ER and mitochondria, and emerging data indicate their vital role in the regulation of fundamental physiological processes, chief among them mitochondria bioenergetics, proteostasis, cell death, and autophagy. Moreover, and perhaps not surprisingly, it has become clear that signaling events regulated at the ER?mitochondria intersection regulate key processes in oncogenesis and in the response of cancer cells to therapeutics. ER?mitochondria appositions have been shown to dynamically recruit oncogenes and tumor suppressors, modulating their activity and protein complex formation, adapt the bioenergetic demand of cancer cells and to regulate cell death pathways and redox signaling in cancer cells. In this review, we discuss some emerging players of the ER?mitochondria contact sites in mammalian cells, the key processes they regulate and recent evidence highlighting the role of MAMs in shaping cell-autonomous and non-autonomous signals that regulate cancer growth. äMitochondria-Associated Membranes As Networking Platforms and Regulato(epmc).pdf DeepDyve Pubget Overpricing