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10.4049/jimmunol.1601193 http://scihub22266oqcxt.onion/10.4049/jimmunol.1601193 C5557026!5557026!28607116     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Immunol 2017 ; 199 (2): 633-42 Nephropedia Template TP {{tp|p=28607116|t=2017. Elimination of Babesia microti is dependent on intraerythroytic killing and CD4+ T cells |pdf=|usr=}}{{28607116}} gab.com Text Elimination of Babesia microti is dependent on intraerythroytic killing and CD4+ T cells JO: J Immunol http://www.kidney.de/pget.php?&tw=1&pmid=28607116 #FOAvip Babesiosis is a tick-borne zoonosis caused by protozoans of the genus Babesia; apicomplexan parasites that replicate within erythrocytes. However, unlike related Plasmodium species, the pathogenesis of Babesia infection remains poorly understood. The primary etiological agent of babesiosis in the US is B. microti. In healthy individuals, tick transmitted infection with Babesia causes no specific clinical manifestations, with many having no symptoms at all. However, even in asymptomatic people, a Babesia carriage state can be established that can last up to a year or more. Current blood bank screening methods do not identify infected donors, and Babesia parasites survive blood banking procedures and storage. Thus, Babesia can also be transmitted by infected blood, and is currently the number one cause of reportable transfusion transmitted infection in the US. Despite a significant impact on human health, B. microti remains understudied. Here, we evaluated the course of Babesia infection in three different strains of mice, C57BL/6J, BALB/cJ and C3H-HeJ, and examined the contribution of multiple immune parameters including: toll-like receptors, B cells, CD4+ cells, IFN-?, and nitric oxide on the level of parasitemia and parasite clearance during acute babesiosis. We found that B. microti reaches high parasitemia levels during the first week of infection in all three mice strains before resolving spontaneously. Our results indicate that resolution of babesiosis requires CD4 T cells and a novel mechanism of parasite killing within infected erythrocytes. Twit Text FOAVip Elimination of Babesia microti is dependent on intraerythroytic killing and CD4+ T cells ????J Immunol http://www.kidney.de/pget.php?&tw=1&pmid=28607116 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28607116 #FOAvip English Wikipedia <ref>{{Cite pmid|28607116}}</ref> Elimination of Babesia microti is dependent on intraerythroytic killing and CD4+ T cells #MMPMID28607116 Skariah S; Arnaboldi P; Dattwyler RJ; Sultan AA; Gaylets C; Walwyn O; Mullhall H; Wu Z; Dargham SR; Mordue DG J Immunol 2017[Jul]; 199 (2): 633-42 PMID28607116show ga Babesiosis is a tick-borne zoonosis caused by protozoans of the genus Babesia; apicomplexan parasites that replicate within erythrocytes. However, unlike related Plasmodium species, the pathogenesis of Babesia infection remains poorly understood. The primary etiological agent of babesiosis in the US is B. microti. In healthy individuals, tick transmitted infection with Babesia causes no specific clinical manifestations, with many having no symptoms at all. However, even in asymptomatic people, a Babesia carriage state can be established that can last up to a year or more. Current blood bank screening methods do not identify infected donors, and Babesia parasites survive blood banking procedures and storage. Thus, Babesia can also be transmitted by infected blood, and is currently the number one cause of reportable transfusion transmitted infection in the US. Despite a significant impact on human health, B. microti remains understudied. Here, we evaluated the course of Babesia infection in three different strains of mice, C57BL/6J, BALB/cJ and C3H-HeJ, and examined the contribution of multiple immune parameters including: toll-like receptors, B cells, CD4+ cells, IFN-?, and nitric oxide on the level of parasitemia and parasite clearance during acute babesiosis. We found that B. microti reaches high parasitemia levels during the first week of infection in all three mice strains before resolving spontaneously. Our results indicate that resolution of babesiosis requires CD4 T cells and a novel mechanism of parasite killing within infected erythrocytes. äElimination of Babesia microti is dependent on intraerythroytic killin(epmc).pdf DeepDyve Pubget Overpricing