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10.1152/ajpheart.00135.2017 http://scihub22266oqcxt.onion/10.1152/ajpheart.00135.2017 C5538862!5538862!28526707     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 265.2 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Am+J+Physiol+Heart+Circ+Physiol 2017 ; 313 (1): H66-71 Nephropedia Template TP {{tp|p=28526707|t=2017. S-nitrosylation of VASP at cysteine 64 mediates the inflammation-stimulated increase in microvascular permeability |pdf=|usr=}}{{28526707}} gab.com Text S-nitrosylation of VASP at cysteine 64 mediates the inflammation-stimulated increase in microvascular permeability JO: Am J Physiol Heart Circ Physiol http://www.kidney.de/pget.php?&tw=1&pmid=28526707 #FOAvip Here, we demonstrate that S-nitrosylation of vasodilator-stimulated phosphoprotein (VASP) on C64 is a mechanism for the onset of platelet-activating factor-induced hyperpermeability. Our results reveal a dual role of VASP in endothelial permeability. In addition to its well-documented function in barrier integrity, we show that S-nitrosylation of VASP contributes to the onset of endothelial permeability. Twit Text FOAVip S-nitrosylation of VASP at cysteine 64 mediates the inflammation-stimulated increase in microvascular permeability ????Am J Physiol Heart Circ Physiol http://www.kidney.de/pget.php?&tw=1&pmid=28526707 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28526707 #FOAvip English Wikipedia <ref>{{Cite pmid|28526707}}</ref> S-nitrosylation of VASP at cysteine 64 mediates the inflammation-stimulated increase in microvascular permeability #MMPMID28526707 Zamorano P; Marín N; Córdova F; Aguilar A; Meininger C; Boric MP; Golenhofen N; Contreras JE; Sarmiento J; Durán WN; Sánchez FA Am J Physiol Heart Circ Physiol 2017[Jul]; 313 (1): H66-71 PMID28526707show ga Here, we demonstrate that S-nitrosylation of vasodilator-stimulated phosphoprotein (VASP) on C64 is a mechanism for the onset of platelet-activating factor-induced hyperpermeability. Our results reveal a dual role of VASP in endothelial permeability. In addition to its well-documented function in barrier integrity, we show that S-nitrosylation of VASP contributes to the onset of endothelial permeability. äS-nitrosylation of VASP at cysteine 64 mediates the inflammation-stimu(epmc).pdf DeepDyve Pubget Overpricing