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10.1161/HYPERTENSIONAHA.116.08316 http://scihub22266oqcxt.onion/10.1161/HYPERTENSIONAHA.116.08316 C5509064!5509064!28137982     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28137982&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Hypertension 2017 ; 69 (3): 387-95 Nephropedia Template TP {{tp|p=28137982|t=2017. FATE OF RENIN CELLS DURING DEVELOPMENT AND DISEASE |pdf=|usr=}}{{28137982}} gab.com Text FATE OF RENIN CELLS DURING DEVELOPMENT AND DISEASE JO: Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=28137982 #FOAvip Although renin cells are crucial for blood pressure homeostasis, little is known about their nature. We now know that renin cells are precursors that appear early and in multiple tissues during embryonic development. They participate in morphogenetic events, vascular development and injury, tissue repair and regeneration. When confronted to a homeostatic threat, renin cell descendants have the capability to switch the renin gene on or off. This poorly understood switch or molecular memory enables the organism to maintain constancy of the internal milieu and tissue perfussion. Here, we discuss briefly the major events that govern the acquisition and maintenace of renin cell identity and how manipulations that alter the fate of renin cells can lead to serious disease. We also advance the concept that renin cells are at the center of an ancestral sytem of defense linking the endocrine, the immune and repair responses of the organism. Twit Text FOAVip FATE OF RENIN CELLS DURING DEVELOPMENT AND DISEASE ????Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=28137982 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28137982 #FOAvip English Wikipedia <ref>{{Cite pmid|28137982}}</ref> FATE OF RENIN CELLS DURING DEVELOPMENT AND DISEASE #MMPMID28137982 Gomez RA Hypertension 2017[Mar]; 69 (3): 387-95 PMID28137982show ga Although renin cells are crucial for blood pressure homeostasis, little is known about their nature. We now know that renin cells are precursors that appear early and in multiple tissues during embryonic development. They participate in morphogenetic events, vascular development and injury, tissue repair and regeneration. When confronted to a homeostatic threat, renin cell descendants have the capability to switch the renin gene on or off. This poorly understood switch or molecular memory enables the organism to maintain constancy of the internal milieu and tissue perfussion. Here, we discuss briefly the major events that govern the acquisition and maintenace of renin cell identity and how manipulations that alter the fate of renin cells can lead to serious disease. We also advance the concept that renin cells are at the center of an ancestral sytem of defense linking the endocrine, the immune and repair responses of the organism. äFATE OF RENIN CELLS DURING DEVELOPMENT AND DISEASE (epmc).pdf DeepDyve Pubget Overpricing