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10.1152/ajprenal.00488.2015 http://scihub22266oqcxt.onion/10.1152/ajprenal.00488.2015 C5504458!5504458!26764204     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26764204.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Am+J+Physiol+Renal+Physiol 2016 ; 310 (8): F785-95 Nephropedia Template TP {{tp|p=26764204|t=2016. Oxalate-induced chronic kidney disease with its uremic and cardiovascular complications in C57BL/6 mice |pdf=|usr=}}{{26764204}} gab.com Text Oxalate-induced chronic kidney disease with its uremic and cardiovascular complications in C57BL/6 mice JO: Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=26764204 #FOAvip Chronic kidney disease (CKD) research is limited by the lack of convenient inducible models mimicking human CKD and its complications in experimental animals. We demonstrate that a soluble oxalate-rich diet induces stable stages of CKD in male and female C57BL/6 mice. Renal histology is characterized by tubular damage, remnant atubular glomeruli, interstitial inflammation, and fibrosis, with the extent of tissue involvement depending on the duration of oxalate feeding. Expression profiling of markers and magnetic resonance imaging findings established to reflect inflammation and fibrosis parallel the histological changes. Within 3 wk, the mice reproducibly develop normochromic anemia, metabolic acidosis, hyperkalemia, FGF23 activation, hyperphosphatemia, and hyperparathyroidism. In addition, the model is characterized by profound arterial hypertension as well as cardiac fibrosis that persist following the switch to a control diet. Together, this new model of inducible CKD overcomes a number of previous experimental limitations and should serve useful in research related to CKD and its complications. Twit Text FOAVip Oxalate-induced chronic kidney disease with its uremic and cardiovascular complications in C57BL/6 mice ????Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=26764204 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26764204 #FOAvip English Wikipedia <ref>{{Cite pmid|26764204}}</ref> Oxalate-induced chronic kidney disease with its uremic and cardiovascular complications in C57BL/6 mice #MMPMID26764204 Mulay SR; Eberhard JN; Pfann V; Marschner JA; Darisipudi MN; Daniel C; Romoli S; Desai J; Grigorescu M; Kumar SV; Rathkolb B; Wolf E; Hrab? de Angelis M; Bäuerle T; Dietel B; Wagner CA; Amann K; Eckardt KU; Aronson PS; Anders HJ; Knauf F Am J Physiol Renal Physiol 2016[Apr]; 310 (8): F785-95 PMID26764204show ga Chronic kidney disease (CKD) research is limited by the lack of convenient inducible models mimicking human CKD and its complications in experimental animals. We demonstrate that a soluble oxalate-rich diet induces stable stages of CKD in male and female C57BL/6 mice. Renal histology is characterized by tubular damage, remnant atubular glomeruli, interstitial inflammation, and fibrosis, with the extent of tissue involvement depending on the duration of oxalate feeding. Expression profiling of markers and magnetic resonance imaging findings established to reflect inflammation and fibrosis parallel the histological changes. Within 3 wk, the mice reproducibly develop normochromic anemia, metabolic acidosis, hyperkalemia, FGF23 activation, hyperphosphatemia, and hyperparathyroidism. In addition, the model is characterized by profound arterial hypertension as well as cardiac fibrosis that persist following the switch to a control diet. Together, this new model of inducible CKD overcomes a number of previous experimental limitations and should serve useful in research related to CKD and its complications. äOxalate-induced chronic kidney disease with its uremic and cardiovascu(epmc).pdf DeepDyve Pubget Overpricing