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10.1083/jcb.201610113 http://scihub22266oqcxt.onion/10.1083/jcb.201610113 C5496614!5496614!28566325     free     free     free Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 227.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Cell+Biol 2017 ; 216 (7): 1949-57 Nephropedia Template TP {{tp|p=28566325|t=2017. Persistent mTORC1 signaling in cell senescence results from defects in amino acid and growth factor sensing |pdf=|usr=}}{{28566325}} gab.com Text Persistent mTORC1 signaling in cell senescence results from defects in amino acid and growth factor sensing JO: J Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=28566325 #FOAvip It is unclear how mTORC1 signaling differs in senescent and young cells. Carroll et al. show that senescence leads to constitutive mTORC1 activation and resistance to serum and amino acid starvation. This is associated with elevated autophagy, depolarization of cell plasma membrane, and primary cilia defects. Twit Text FOAVip Persistent mTORC1 signaling in cell senescence results from defects in amino acid and growth factor sensing ????J Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=28566325 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28566325 #FOAvip English Wikipedia <ref>{{Cite pmid|28566325}}</ref> Persistent mTORC1 signaling in cell senescence results from defects in amino acid and growth factor sensing #MMPMID28566325 Carroll B; Nelson G; Rabanal-Ruiz Y; Kucheryavenko O; Dunhill-Turner NA; Chesterman CC; Zahari Q; Zhang T; Conduit SE; Mitchell CA; Maddocks OD; Lovat P; von Zglinicki T; Korolchuk VI J Cell Biol 2017[Jul]; 216 (7): 1949-57 PMID28566325show ga It is unclear how mTORC1 signaling differs in senescent and young cells. Carroll et al. show that senescence leads to constitutive mTORC1 activation and resistance to serum and amino acid starvation. This is associated with elevated autophagy, depolarization of cell plasma membrane, and primary cilia defects. äPersistent mTORC1 signaling in cell senescence results from defects in(epmc).pdf DeepDyve Pubget Overpricing