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10.1002/1878-0261.12083 http://scihub22266oqcxt.onion/10.1002/1878-0261.12083 C5496498!5496498!28548345     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28548345&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Mol+Oncol 2017 ; 11 (7): 755-69 Nephropedia Template TP {{tp|p=28548345|t=2017. EMT and MET: necessary or permissive for metastasis?|pdf=|usr=}}{{28548345}} gab.com Text EMT and MET: necessary or permissive for metastasis JO: Mol Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28548345 #FOAvip Epithelial?to?mesenchymal transition (EMT) and its reverse mesenchymal?to?epithelial transition (MET) have been suggested to play crucial roles in metastatic dissemination of carcinomas. These phenotypic transitions between states are not binary. Instead, carcinoma cells often exhibit a spectrum of epithelial/mesenchymal phenotype(s). While epithelial/mesenchymal plasticity has been observed preclinically and clinically, whether any of these phenotypic transitions are indispensable for metastatic outgrowth remains an unanswered question. Here, we focus on epithelial/mesenchymal plasticity in metastatic dissemination and propose alternative mechanisms for successful dissemination and metastases beyond the traditional EMT/MET view. We highlight multiple hypotheses that can help reconcile conflicting observations, and outline the next set of key questions that can offer valuable insights into mechanisms of metastasis in multiple tumor models. Twit Text FOAVip EMT and MET: necessary or permissive for metastasis ????Mol Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28548345 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28548345 #FOAvip English Wikipedia <ref>{{Cite pmid|28548345}}</ref> EMT and MET: necessary or permissive for metastasis? #MMPMID28548345 Jolly MK; Ware KE; Gilja S; Somarelli JA; Levine H Mol Oncol 2017[Jul]; 11 (7): 755-69 PMID28548345show ga Epithelial?to?mesenchymal transition (EMT) and its reverse mesenchymal?to?epithelial transition (MET) have been suggested to play crucial roles in metastatic dissemination of carcinomas. These phenotypic transitions between states are not binary. Instead, carcinoma cells often exhibit a spectrum of epithelial/mesenchymal phenotype(s). While epithelial/mesenchymal plasticity has been observed preclinically and clinically, whether any of these phenotypic transitions are indispensable for metastatic outgrowth remains an unanswered question. Here, we focus on epithelial/mesenchymal plasticity in metastatic dissemination and propose alternative mechanisms for successful dissemination and metastases beyond the traditional EMT/MET view. We highlight multiple hypotheses that can help reconcile conflicting observations, and outline the next set of key questions that can offer valuable insights into mechanisms of metastasis in multiple tumor models. äEMT and MET: necessary or permissive for metastasis?(epmc).pdf DeepDyve Pubget Overpricing