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10.5607/en.2017.26.3.172 http://scihub22266oqcxt.onion/10.5607/en.2017.26.3.172 C5491586!5491586!28680303     free     free     free Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Exp+Neurobiol 2017 ; 26 (3): 172-7 Nephropedia Template TP {{tp|p=28680303|t=2017. An Autopsy Proven Child Onset Chronic Traumatic Encephalopathy |pdf=|usr=}}{{28680303}} gab.com Text An Autopsy Proven Child Onset Chronic Traumatic Encephalopathy JO: Exp Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=28680303 #FOAvip Here we present an autopsy case of chronic traumatic encephalopathy (CTE) in a 36-year-old man. He had a history of febrile seizures at the age of four and was severely demented at age 10 when he was admitted to a mental hospital. He had suffered repetitive self-harm, such as frequent banging of the head on the wall in his hospital record, but he had no clear history between the ages of four and ten. Autopsy revealed global cerebral atrophy, including the basal ganglia, thalamus, hippocampus, amygdala, mammilary bodies and lateral geniculate bodies. This case showed typical pathological features of CTE. Phosphorylated tau (p-tau)-positive neurofibrillary tangles (NFTs) and neuropil threads (NT) we are widely distributed in the brain, especially in the depth of the cerebral sulci. NFT and NT were also found in the basal ganglia, thalamus, amygdala and brainstem. Scanty ?-amyloid deposits were found in the motor and sensory cortices, but ?-synuclein was completely negative in the brain. This example showed that CTE can occur in young ages and that even children can experience CTE dementia. Twit Text FOAVip An Autopsy Proven Child Onset Chronic Traumatic Encephalopathy ????Exp Neurobiol http://www.kidney.de/pget.php?&tw=1&pmid=28680303 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28680303 #FOAvip English Wikipedia <ref>{{Cite pmid|28680303}}</ref> An Autopsy Proven Child Onset Chronic Traumatic Encephalopathy #MMPMID28680303 Lee K; Kim SI; Lee Y; Won JK; Park SH Exp Neurobiol 2017[Jun]; 26 (3): 172-7 PMID28680303show ga Here we present an autopsy case of chronic traumatic encephalopathy (CTE) in a 36-year-old man. He had a history of febrile seizures at the age of four and was severely demented at age 10 when he was admitted to a mental hospital. He had suffered repetitive self-harm, such as frequent banging of the head on the wall in his hospital record, but he had no clear history between the ages of four and ten. Autopsy revealed global cerebral atrophy, including the basal ganglia, thalamus, hippocampus, amygdala, mammilary bodies and lateral geniculate bodies. This case showed typical pathological features of CTE. Phosphorylated tau (p-tau)-positive neurofibrillary tangles (NFTs) and neuropil threads (NT) we are widely distributed in the brain, especially in the depth of the cerebral sulci. NFT and NT were also found in the basal ganglia, thalamus, amygdala and brainstem. Scanty ?-amyloid deposits were found in the motor and sensory cortices, but ?-synuclein was completely negative in the brain. This example showed that CTE can occur in young ages and that even children can experience CTE dementia. äAn Autopsy Proven Child Onset Chronic Traumatic Encephalopathy (epmc).pdf DeepDyve Pubget Overpricing