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http://scihub22266oqcxt.onion/ C5489895!5489895 !28670383     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28670383 &cmd=llinks): Failed to open stream: HTTP request failed! 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MiR-155 up-regulated by TGF-? promotes epithelial-mesenchymal transition, invasion and metastasis of human hepatocellular carcinoma cells in vitro |pdf=|usr=}}{{28670383 }} gab.com Text MiR-155 up-regulated by TGF- promotes epithelial-mesenchymal transition, invasion and metastasis of human hepatocellular carcinoma cells in vitro JO: Am J Transl Res http://www.kidney.de/pget.php?&tw=1&pmid=28670383 #FOAvip It has previously been reported that microRNA (miR)-155 is linked to the recurrence and prognosis of hepatocellular carcinoma (HCC) following liver transplantation. However, the role of miR-155 in the invasion and metastasis of HCC cells remains largely unclear. The aim of this study was to investigate the expression of miR-155 in HCC cells and its role in the invasion and migration of HCC cells in vitro. We found that the level of expression of miR-155 in HCC tissues and cells was significantly increased compared with non-tumorous adjacent tissues. Further study revealed that recombinant human transforming growth factor-? (TGF-?1) up-regulated the expression of miR-155 in HCC cells in vitro. Further, the overexpression of miR-155 in HCC cell line Huh-7 led to increased levels of cell invasion and migration compared with untreated control Huh-7 cells. MiR-155-overexpressed Huh-7 cells also exhibited altered levels of expression of certain cellular adhesion molecules related to epithelial-mesenchymal transition (EMT), including low levels of CDH1 and higher levels of FN1, SNAI1 and ZEB1, compared with control Huh-7 cells. Moreover, it was found that the overexpression of miR-155 and of TGF-?1 protein decreased the expression of E-Cadherin and increased the expression of Vimentin in Huh-7 cells. These results indicate that an increased level of miR-155 in HCC cells, possibly due to stimulation by TGF-?1, accelerates the process of EMT, promotes cellular invasion and migration in vitro, and thereby further promotes the progression of HCC. Twit Text FOAVip MiR-155 up-regulated by TGF- promotes epithelial-mesenchymal transition, invasion and metastasis of human hepatocellular carcinoma cells in vitro ????Am J Transl Res http://www.kidney.de/pget.php?&tw=1&pmid=28670383 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28670383 #FOAvip English Wikipedia <ref>{{Cite pmid|28670383 }}</ref> MiR-155 up-regulated by TGF-? promotes epithelial-mesenchymal transition, invasion and metastasis of human hepatocellular carcinoma cells in vitro #MMPMID28670383 Li DP ; Fan J ; Wu YJ ; Xie YF ; Zha JM ; Zhou XM Am J Transl Res 2017[]; 9 (6 ): 2956-2965 PMID28670383 show ga It has previously been reported that microRNA (miR)-155 is linked to the recurrence and prognosis of hepatocellular carcinoma (HCC) following liver transplantation. However, the role of miR-155 in the invasion and metastasis of HCC cells remains largely unclear. The aim of this study was to investigate the expression of miR-155 in HCC cells and its role in the invasion and migration of HCC cells in vitro. We found that the level of expression of miR-155 in HCC tissues and cells was significantly increased compared with non-tumorous adjacent tissues. Further study revealed that recombinant human transforming growth factor-? (TGF-?1) up-regulated the expression of miR-155 in HCC cells in vitro. Further, the overexpression of miR-155 in HCC cell line Huh-7 led to increased levels of cell invasion and migration compared with untreated control Huh-7 cells. MiR-155-overexpressed Huh-7 cells also exhibited altered levels of expression of certain cellular adhesion molecules related to epithelial-mesenchymal transition (EMT), including low levels of CDH1 and higher levels of FN1, SNAI1 and ZEB1, compared with control Huh-7 cells. Moreover, it was found that the overexpression of miR-155 and of TGF-?1 protein decreased the expression of E-Cadherin and increased the expression of Vimentin in Huh-7 cells. These results indicate that an increased level of miR-155 in HCC cells, possibly due to stimulation by TGF-?1, accelerates the process of EMT, promotes cellular invasion and migration in vitro, and thereby further promotes the progression of HCC. äMiR-155 up-regulated by TGF-? promotes epithelial-mesenchymal transiti(epmc).pdf DeepDyve Pubget Overpricing