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10.3389/fped.2017.00151 http://scihub22266oqcxt.onion/10.3389/fped.2017.00151 C5489607!5489607!28706894     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Pediatr 2017 ; 5 (ä): ä Nephropedia Template TP {{tp|p=28706894|t=2017. Developmental Origins and Nephron Endowment in Hypertension |pdf=|usr=}}{{28706894}} gab.com Text Developmental Origins and Nephron Endowment in Hypertension JO: Front Pediatr http://www.kidney.de/pget.php?&tw=1&pmid=28706894 #FOAvip Primary hypertension continues to be one of the main risk factors for cardiovascular disease worldwide. A stable intrauterine environment is critical for the future development and health of the fetus. The developing kidney has been found to be especially vulnerable during this time period, and epidemiological studies have demonstrated that an adverse in utero environment is associated with an increased risk of hypertension and chronic kidney disease. Macro- and micronutrient deficiencies as well as exposure to tobacco, alcohol, and certain medications during gestation have been shown to negatively impact nephrogenesis and reduce one?s nephron number. In 1988, Brenner et al. put forth the controversial hypothesis that a reduced nephron complement is a risk factor for hypertension and chronic kidney disease in adulthood. Since then numerous animal and human studies have confirmed this relationship demonstrating that there is an inverse association between blood pressure and nephron number. As our understanding of the developmental programming of hypertension and other non-communicable diseases improves, more effective preventive health measures can be developed in the future. Twit Text FOAVip Developmental Origins and Nephron Endowment in Hypertension ????Front Pediatr http://www.kidney.de/pget.php?&tw=1&pmid=28706894 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28706894 #FOAvip English Wikipedia <ref>{{Cite pmid|28706894}}</ref> Developmental Origins and Nephron Endowment in Hypertension #MMPMID28706894 Gurusinghe S; Tambay A; Sethna CB Front Pediatr 2017[]; 5 (ä): ä PMID28706894show ga Primary hypertension continues to be one of the main risk factors for cardiovascular disease worldwide. A stable intrauterine environment is critical for the future development and health of the fetus. The developing kidney has been found to be especially vulnerable during this time period, and epidemiological studies have demonstrated that an adverse in utero environment is associated with an increased risk of hypertension and chronic kidney disease. Macro- and micronutrient deficiencies as well as exposure to tobacco, alcohol, and certain medications during gestation have been shown to negatively impact nephrogenesis and reduce one?s nephron number. In 1988, Brenner et al. put forth the controversial hypothesis that a reduced nephron complement is a risk factor for hypertension and chronic kidney disease in adulthood. Since then numerous animal and human studies have confirmed this relationship demonstrating that there is an inverse association between blood pressure and nephron number. As our understanding of the developmental programming of hypertension and other non-communicable diseases improves, more effective preventive health measures can be developed in the future. äDevelopmental Origins and Nephron Endowment in Hypertension (epmc).pdf DeepDyve Pubget Overpricing