Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.jneuroim.2017.03.014 http://scihub22266oqcxt.onion/10.1016/j.jneuroim.2017.03.014 C5489071!5489071!28335992     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Neuroimmunol 2017 ; 308 (ä): 112-7 Nephropedia Template TP {{tp|p=28335992|t=2017. Varicella zoster virus vasculopathy: the expanding clinical spectrum and pathogenesis |pdf=|usr=}}{{28335992}} gab.com Text Varicella zoster virus vasculopathy: the expanding clinical spectrum and pathogenesis JO: J Neuroimmunol http://www.kidney.de/pget.php?&tw=1&pmid=28335992 #FOAvip Varicella zoster virus (VZV) is a ubiquitous, human alphaherpesvirus that produces varicella on primary infection then becomes latent in ganglionic neurons along the entire neuraxis. In elderly and immunocompromised individuals, VZV reactivates and travels along nerve fibers peripherally resulting in zoster. However, VZV can also spread centrally and infect cerebral and extracranial arteries (VZV vasculopathy) to produce transient ischemic attacks, stroke, aneurysm, sinus thrombosis and giant cell arteritis, as well as granulomatous aortitis. The mechanisms of virus-induced pathological vascular remodeling are not fully elucidated; however, recent studies suggest that inflammation and dysregulation of programmed death ligand-1 play a significant role. Twit Text FOAVip Varicella zoster virus vasculopathy: the expanding clinical spectrum and pathogenesis ????J Neuroimmunol http://www.kidney.de/pget.php?&tw=1&pmid=28335992 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28335992 #FOAvip English Wikipedia <ref>{{Cite pmid|28335992}}</ref> Varicella zoster virus vasculopathy: the expanding clinical spectrum and pathogenesis #MMPMID28335992 Nagel MA; Jones D; Wyborny A J Neuroimmunol 2017[Jul]; 308 (ä): 112-7 PMID28335992show ga Varicella zoster virus (VZV) is a ubiquitous, human alphaherpesvirus that produces varicella on primary infection then becomes latent in ganglionic neurons along the entire neuraxis. In elderly and immunocompromised individuals, VZV reactivates and travels along nerve fibers peripherally resulting in zoster. However, VZV can also spread centrally and infect cerebral and extracranial arteries (VZV vasculopathy) to produce transient ischemic attacks, stroke, aneurysm, sinus thrombosis and giant cell arteritis, as well as granulomatous aortitis. The mechanisms of virus-induced pathological vascular remodeling are not fully elucidated; however, recent studies suggest that inflammation and dysregulation of programmed death ligand-1 play a significant role. äVaricella zoster virus vasculopathy: the expanding clinical spectrum a(epmc).pdf DeepDyve Pubget Overpricing