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10.1126/science.aaf8924 http://scihub22266oqcxt.onion/10.1126/science.aaf8924 C5488328!5488328!27811267     free     free     free Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Science 2016 ; 354 (6312): 572-7 Nephropedia Template TP {{tp|p=27811267|t=2016. Pain Regulation by Non-neuronal Cells and Inflammation |pdf=|usr=}}{{27811267}} gab.com Text Pain Regulation by Non-neuronal Cells and Inflammation JO: Science http://www.kidney.de/pget.php?&tw=1&pmid=27811267 #FOAvip Acute pain is protective and a cardinal feature of inflammation. Chronic pain after arthritis, nerve injury, cancer, and chemotherapy is associated with chronic neuroinflammation, a local inflammation in the peripheral or central nervous system. Accumulating evidence suggests that non-neuronal cells such as immune cells, glial cells, keratinocytes, cancer cells, and stem cells, play active roles in the pathogenesis and resolution of pain. We review how non-neuronal cells interact with nociceptive neurons by secreting neuroactive signaling molecules that modulate pain. Recent studies also suggest bacterial infections regulate pain through direct actions on sensory neurons, and specific receptors are present in nociceptors to detect danger signals from infections. We also discuss new therapeutic strategies to control neuroinflammation for the prevention and treatment of chronic pain. Twit Text FOAVip Pain Regulation by Non-neuronal Cells and Inflammation ????Science http://www.kidney.de/pget.php?&tw=1&pmid=27811267 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27811267 #FOAvip English Wikipedia <ref>{{Cite pmid|27811267}}</ref> Pain Regulation by Non-neuronal Cells and Inflammation #MMPMID27811267 Ji RR; Chamessian A; Zhang YQ Science 2016[Nov]; 354 (6312): 572-7 PMID27811267show ga Acute pain is protective and a cardinal feature of inflammation. Chronic pain after arthritis, nerve injury, cancer, and chemotherapy is associated with chronic neuroinflammation, a local inflammation in the peripheral or central nervous system. Accumulating evidence suggests that non-neuronal cells such as immune cells, glial cells, keratinocytes, cancer cells, and stem cells, play active roles in the pathogenesis and resolution of pain. We review how non-neuronal cells interact with nociceptive neurons by secreting neuroactive signaling molecules that modulate pain. Recent studies also suggest bacterial infections regulate pain through direct actions on sensory neurons, and specific receptors are present in nociceptors to detect danger signals from infections. We also discuss new therapeutic strategies to control neuroinflammation for the prevention and treatment of chronic pain. äPain Regulation by Non-neuronal Cells and Inflammation (epmc).pdf DeepDyve Pubget Overpricing