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10.1051/bmdcn/2017070201 http://scihub22266oqcxt.onion/10.1051/bmdcn/2017070201 C5479440!5479440!28612706     free     free     free       Biomedicine+(Taipei) ä ; 7 (2): ä Nephropedia Template TP {{tp|p=28612706|t=ä. Autophagy and its link to type II diabetes mellitus |pdf=|usr=}}{{28612706}} gab.com Text Autophagy and its link to type II diabetes mellitus JO: Biomedicine (Taipei) http://www.kidney.de/pget.php?&tw=1&pmid=28612706 #FOAvip Autophagy, a double-edged sword for cell survival, is the research object on 2016 Nobel Prize in Physiology or Medicine. Autophagy is a molecular mechanism for maintaining cellular physiology and promoting survival. Defects in autophagy lead to the etiology of many diseases, including diabetes mellitus (DM), cancer, neurodegeneration, infection disease and aging. DM is a metabolic and chronic disorder and has a higher prevalence in the world as well as in Taiwan. The character of diabetes mellitus is hyperglycemia resulting from defects in insulin secretion, insulin action, or both. Type 2 diabetes mellitus (T2DM) is characterized by insulin resistance and failure of producing insulin on pancreatic beta cells. In T2DM, autophagy is not only providing nutrients to maintain cellular energy during fasting, but also removes damaged organelles, lipids and miss-folded proteins. In addition, autophagy plays an important role in pancreatic beta cell dysfunction and insulin resistance. In this review, we summarize the roles of autophagy in T2DM. Twit Text FOAVip Autophagy and its link to type II diabetes mellitus ????Biomedicine (Taipei) http://www.kidney.de/pget.php?&tw=1&pmid=28612706 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28612706 #FOAvip English Wikipedia <ref>{{Cite pmid|28612706}}</ref> Autophagy and its link to type II diabetes mellitus #MMPMID28612706 Yang JS; Lu CC; Kuo SC; Hsu YM; Tsai SC; Chen SY; Chen YT; Lin YJ; Huang YC; Chen CJ; Lin WD; Liao WL; Lin WY; Liu YH; Sheu JC; Tsai FJ Biomedicine (Taipei) ä[]; 7 (2): ä PMID28612706show ga Autophagy, a double-edged sword for cell survival, is the research object on 2016 Nobel Prize in Physiology or Medicine. Autophagy is a molecular mechanism for maintaining cellular physiology and promoting survival. Defects in autophagy lead to the etiology of many diseases, including diabetes mellitus (DM), cancer, neurodegeneration, infection disease and aging. DM is a metabolic and chronic disorder and has a higher prevalence in the world as well as in Taiwan. The character of diabetes mellitus is hyperglycemia resulting from defects in insulin secretion, insulin action, or both. Type 2 diabetes mellitus (T2DM) is characterized by insulin resistance and failure of producing insulin on pancreatic beta cells. In T2DM, autophagy is not only providing nutrients to maintain cellular energy during fasting, but also removes damaged organelles, lipids and miss-folded proteins. In addition, autophagy plays an important role in pancreatic beta cell dysfunction and insulin resistance. In this review, we summarize the roles of autophagy in T2DM. äAutophagy and its link to type II diabetes mellitus (epmc).pdf DeepDyve Pubget Overpricing