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10.1186/s13059-017-1250-y http://scihub22266oqcxt.onion/10.1186/s13059-017-1250-y C5477265!5477265!28629478     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Genome+Biol 2017 ; 18 (ä): ä Nephropedia Template TP {{tp|p=28629478|t=2017. Genetic?epigenetic interactions in cis: a major focus in the post-GWAS era |pdf=|usr=}}{{28629478}} gab.com Text Genetic epigenetic interactions in cis: a major focus in the post-GWAS era JO: Genome Biol http://www.kidney.de/pget.php?&tw=1&pmid=28629478 #FOAvip Studies on genetic?epigenetic interactions, including the mapping of methylation quantitative trait loci (mQTLs) and haplotype-dependent allele-specific DNA methylation (hap-ASM), have become a major focus in the post-genome-wide-association-study (GWAS) era. Such maps can nominate regulatory sequence variants that underlie GWAS signals for common diseases, ranging from neuropsychiatric disorders to cancers. Conversely, mQTLs need to be filtered out when searching for non-genetic effects in epigenome-wide association studies (EWAS). Sequence variants in CCCTC-binding factor (CTCF) and transcription factor binding sites have been mechanistically linked to mQTLs and hap-ASM. Identifying these sites can point to disease-associated transcriptional pathways, with implications for targeted treatment and prevention. Twit Text FOAVip Genetic epigenetic interactions in cis: a major focus in the post-GWAS era ????Genome Biol http://www.kidney.de/pget.php?&tw=1&pmid=28629478 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28629478 #FOAvip English Wikipedia <ref>{{Cite pmid|28629478}}</ref> Genetic?epigenetic interactions in cis: a major focus in the post-GWAS era #MMPMID28629478 Do C; Shearer A; Suzuki M; Terry MB; Gelernter J; Greally JM; Tycko B Genome Biol 2017[]; 18 (ä): ä PMID28629478show ga Studies on genetic?epigenetic interactions, including the mapping of methylation quantitative trait loci (mQTLs) and haplotype-dependent allele-specific DNA methylation (hap-ASM), have become a major focus in the post-genome-wide-association-study (GWAS) era. Such maps can nominate regulatory sequence variants that underlie GWAS signals for common diseases, ranging from neuropsychiatric disorders to cancers. Conversely, mQTLs need to be filtered out when searching for non-genetic effects in epigenome-wide association studies (EWAS). Sequence variants in CCCTC-binding factor (CTCF) and transcription factor binding sites have been mechanistically linked to mQTLs and hap-ASM. Identifying these sites can point to disease-associated transcriptional pathways, with implications for targeted treatment and prevention. äGenetic?epigenetic interactions in cis: a major focus in the post-GWAS(epmc).pdf DeepDyve Pubget Overpricing