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10.2147/JIR.S131051 http://scihub22266oqcxt.onion/10.2147/JIR.S131051 C5476581!5476581!28652797     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Inflamm+Res 2017 ; 10 (ä): 75-81 Nephropedia Template TP {{tp|p=28652797|t=2017. FLLL32, a curcumin analog, ameliorates intestinal injury in necrotizing enterocolitis |pdf=|usr=}}{{28652797}} gab.com Text FLLL32, a curcumin analog, ameliorates intestinal injury in necrotizing enterocolitis JO: J Inflamm Res http://www.kidney.de/pget.php?&tw=1&pmid=28652797 #FOAvip Background: Necrotizing enterocolitis (NEC) is a devastating gastrointestinal disease that primarily affects premature infants. It is characterized by inflammation and leukocyte infiltration that can progress to intestinal necrosis, perforation, systemic inflammatory response, and death. In this study, we examined the effect of FLLL32, a curcumin analog, on an NEC-like neonatal intestinal injury model. Methods: NEC was induced in CD-1 mice pups using the Paneth cell ablation and Klebsiella infection model. Pups were divided into sham, NEC, and NEC + FLLL32 groups. At the end of the experiment, pups were euthanized; whole blood and small intestines were harvested. Intestinal inflammatory cytokine profile, in vivo intestinal permeability using serum fluorescein isothiocyanate-dextran, and histological injury scores were compared between the groups. Results and conclusion: FLLL32-treated pups had lower intestinal injury, improved intestinal permeability, and lower proinflammatory cytokine profiles compared to those in untreated pups with NEC. These results suggest that FLLL32 plays a protective role in NEC. Twit Text FOAVip FLLL32, a curcumin analog, ameliorates intestinal injury in necrotizing enterocolitis ????J Inflamm Res http://www.kidney.de/pget.php?&tw=1&pmid=28652797 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28652797 #FOAvip English Wikipedia <ref>{{Cite pmid|28652797}}</ref> FLLL32, a curcumin analog, ameliorates intestinal injury in necrotizing enterocolitis #MMPMID28652797 Eckert J; Scott B; Lawrence SM; Ihnat M; Chaaban H J Inflamm Res 2017[]; 10 (ä): 75-81 PMID28652797show ga Background: Necrotizing enterocolitis (NEC) is a devastating gastrointestinal disease that primarily affects premature infants. It is characterized by inflammation and leukocyte infiltration that can progress to intestinal necrosis, perforation, systemic inflammatory response, and death. In this study, we examined the effect of FLLL32, a curcumin analog, on an NEC-like neonatal intestinal injury model. Methods: NEC was induced in CD-1 mice pups using the Paneth cell ablation and Klebsiella infection model. Pups were divided into sham, NEC, and NEC + FLLL32 groups. At the end of the experiment, pups were euthanized; whole blood and small intestines were harvested. Intestinal inflammatory cytokine profile, in vivo intestinal permeability using serum fluorescein isothiocyanate-dextran, and histological injury scores were compared between the groups. Results and conclusion: FLLL32-treated pups had lower intestinal injury, improved intestinal permeability, and lower proinflammatory cytokine profiles compared to those in untreated pups with NEC. These results suggest that FLLL32 plays a protective role in NEC. äFLLL32, a curcumin analog, ameliorates intestinal injury in necrotizin(epmc).pdf DeepDyve Pubget Overpricing