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10.1038/ncomms15754 http://scihub22266oqcxt.onion/10.1038/ncomms15754 C5472732!5472732!28598422     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Nat+Commun 2017 ; 8 (ä): ä Nephropedia Template TP {{tp|p=28598422|t=2017. Constriction of the mitochondrial inner compartment is a priming event for mitochondrial division |pdf=|usr=}}{{28598422}} gab.com Text Constriction of the mitochondrial inner compartment is a priming event for mitochondrial division JO: Nat Commun http://www.kidney.de/pget.php?&tw=1&pmid=28598422 #FOAvip Mitochondrial division is critical for the maintenance and regulation of mitochondrial function, quality and distribution. This process is controlled by cytosolic actin-based constriction machinery and dynamin-related protein 1 (Drp1) on mitochondrial outer membrane (OMM). Although mitochondrial physiology, including oxidative phosphorylation, is also important for efficient mitochondrial division, morphological alterations of the mitochondrial inner-membrane (IMM) have not been clearly elucidated. Here we report spontaneous and repetitive constriction of mitochondrial inner compartment (CoMIC) associated with subsequent division in neurons. Although CoMIC is potentiated by inhibition of Drp1 and occurs at the potential division spots contacting the endoplasmic reticulum, it appears on IMM independently of OMM. Intra-mitochondrial influx of Ca2+ induces and potentiates CoMIC, and leads to K+-mediated mitochondrial bulging and depolarization. Synergistically, optic atrophy 1 (Opa1) also regulates CoMIC via controlling Mic60-mediated OMM?IMM tethering. Therefore, we propose that CoMIC is a priming event for efficient mitochondrial division. Twit Text FOAVip Constriction of the mitochondrial inner compartment is a priming event for mitochondrial division ????Nat Commun http://www.kidney.de/pget.php?&tw=1&pmid=28598422 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28598422 #FOAvip English Wikipedia <ref>{{Cite pmid|28598422}}</ref> Constriction of the mitochondrial inner compartment is a priming event for mitochondrial division #MMPMID28598422 Cho B; Cho HM; Jo Y; Kim HD; Song M; Moon C; Kim H; Kim K; Sesaki H; Rhyu IJ; Kim H; Sun W Nat Commun 2017[]; 8 (ä): ä PMID28598422show ga Mitochondrial division is critical for the maintenance and regulation of mitochondrial function, quality and distribution. This process is controlled by cytosolic actin-based constriction machinery and dynamin-related protein 1 (Drp1) on mitochondrial outer membrane (OMM). Although mitochondrial physiology, including oxidative phosphorylation, is also important for efficient mitochondrial division, morphological alterations of the mitochondrial inner-membrane (IMM) have not been clearly elucidated. Here we report spontaneous and repetitive constriction of mitochondrial inner compartment (CoMIC) associated with subsequent division in neurons. Although CoMIC is potentiated by inhibition of Drp1 and occurs at the potential division spots contacting the endoplasmic reticulum, it appears on IMM independently of OMM. Intra-mitochondrial influx of Ca2+ induces and potentiates CoMIC, and leads to K+-mediated mitochondrial bulging and depolarization. Synergistically, optic atrophy 1 (Opa1) also regulates CoMIC via controlling Mic60-mediated OMM?IMM tethering. Therefore, we propose that CoMIC is a priming event for efficient mitochondrial division. äConstriction of the mitochondrial inner compartment is a priming event(epmc).pdf DeepDyve Pubget Overpricing