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10.1371/journal.pone.0179204 http://scihub22266oqcxt.onion/10.1371/journal.pone.0179204 C5466328!5466328!28598982     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       PLoS+One 2017 ; 12 (6): ä Nephropedia Template TP {{tp|p=28598982|t=2017. ?-Mangostin ameliorates hepatic steatosis and insulin resistance by inhibition C-C chemokine receptor 2 |pdf=|usr=}}{{28598982}} gab.com Text -Mangostin ameliorates hepatic steatosis and insulin resistance by inhibition C-C chemokine receptor 2 JO: PLoS One http://www.kidney.de/pget.php?&tw=1&pmid=28598982 #FOAvip Obesity induces various metabolic diseases such as dyslipidemia, nonalcoholic fatty liver disease (NAFLD), and type 2 diabetes. Fat expansion in adipose tissue induces adipose tissue dysfunction and inflammation, insulin resistance, and other metabolic syndromes. ?-Mangostin (?-MG) has been previously studied for its anti-cancer, anti-inflammatory, and antioxidant activities. In this study, we investigated the effects of ?-MG on adipose tissue inflammation and hepatic steatosis. We categorized study animals into four groups: regular diet control mice, RD mice treated with ?-MG, high fat diet-induced obese mice, and HFD mice treated with ?-MG. ?-MG treatment significantly reduced not only the body, liver, and fat weights, but also plasma glucose, insulin, and triglyceride levels in HFD mice. Additionally, adiponectin levels of ?-MG-treated mice were significantly higher than those of control HFD mice. Immunohistochemistry of liver and adipose tissue showed that CD11c expression was reduced in ?-MG fed obese mice. ?-MG treatment of HFD mice down-regulated the adipose-associated inflammatory cytokines and CCR2 in both liver and adipose tissue. Moreover, glucose tolerance and insulin sensitivity were significantly improved in ?-MG fed obese mice. ?-Mangostin ameliorates adipose inflammation and hepatic steatosis in HFD-induced obese mice. Twit Text FOAVip -Mangostin ameliorates hepatic steatosis and insulin resistance by inhibition C-C chemokine receptor 2 ????PLoS One http://www.kidney.de/pget.php?&tw=1&pmid=28598982 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28598982 #FOAvip English Wikipedia <ref>{{Cite pmid|28598982}}</ref> ?-Mangostin ameliorates hepatic steatosis and insulin resistance by inhibition C-C chemokine receptor 2 #MMPMID28598982 Kim HM; Kim YM; Huh JH; Lee ES; Kwon MH; Lee BR; Ko HJ; Chung CH PLoS One 2017[]; 12 (6): ä PMID28598982show ga Obesity induces various metabolic diseases such as dyslipidemia, nonalcoholic fatty liver disease (NAFLD), and type 2 diabetes. Fat expansion in adipose tissue induces adipose tissue dysfunction and inflammation, insulin resistance, and other metabolic syndromes. ?-Mangostin (?-MG) has been previously studied for its anti-cancer, anti-inflammatory, and antioxidant activities. In this study, we investigated the effects of ?-MG on adipose tissue inflammation and hepatic steatosis. We categorized study animals into four groups: regular diet control mice, RD mice treated with ?-MG, high fat diet-induced obese mice, and HFD mice treated with ?-MG. ?-MG treatment significantly reduced not only the body, liver, and fat weights, but also plasma glucose, insulin, and triglyceride levels in HFD mice. Additionally, adiponectin levels of ?-MG-treated mice were significantly higher than those of control HFD mice. Immunohistochemistry of liver and adipose tissue showed that CD11c expression was reduced in ?-MG fed obese mice. ?-MG treatment of HFD mice down-regulated the adipose-associated inflammatory cytokines and CCR2 in both liver and adipose tissue. Moreover, glucose tolerance and insulin sensitivity were significantly improved in ?-MG fed obese mice. ?-Mangostin ameliorates adipose inflammation and hepatic steatosis in HFD-induced obese mice. ä?-Mangostin ameliorates hepatic steatosis and insulin resistance by in(epmc).pdf DeepDyve Pubget Overpricing