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10.1080/23723556.2017.1306610

http://scihub22266oqcxt.onion/10.1080/23723556.2017.1306610
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C5462519!5462519!28616584
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suck abstract from ncbi

pmid28616584      Mol+Cell+Oncol 2017 ; 4 (3): ä
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  • BAK regulates catalase release from peroxisomes #MMPMID28616584
  • Fujiki Y; Miyata N; Mukai S; Okumoto K; Cheng EH
  • Mol Cell Oncol 2017[]; 4 (3): ä PMID28616584show ga
  • Loss of voltage-dependent anion channel 2 (VDAC2) leads to impaired peroxisome biogenesis in mammalian cells. Knockdown of BAK restores peroxisomal biogenesis in VDAC2-deficient cells, where BAK localization shifts from mitochondria to peroxisomes. Moreover, overexpression of BAK activators in wild-type cells permeabilizes peroxisomes in a BAK-dependent manner. Together, BAK most likely regulates peroxisomal membrane permeability.
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