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10.1080/23723556.2017.1304846 http://scihub22266oqcxt.onion/10.1080/23723556.2017.1304846 C5462509!5462509!28616582     free     free     free Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 217.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Mol+Cell+Oncol 2017 ; 4 (3): ä Nephropedia Template TP {{tp|p=28616582|t=2017. A new duet in cancer biology: AMPK the typical and UBE2O the atypical |pdf=|usr=}}{{28616582}} gab.com Text A new duet in cancer biology: AMPK the typical and UBE2O the atypical JO: Mol Cell Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28616582 #FOAvip Ubiquitin-conjugating enzyme E2O (UBE2O) is upregulated in human cancers. We have demonstrated that genetic deletion or pharmacological blockade of UBE2O reduces tumorigenesis through inhibiting the mammalian target of rapamycin complex 1?hypoxia-inducible factor 1-? pathway. Critically, UBE2O targets adenosine monophosphate (AMP)-activated protein kinase-? 2 (AMPK?2) for ubiquitination and degradation. We thus suggest the UBE2O-AMPK?2 axis as a potential therapeutic target for cancer. Twit Text FOAVip A new duet in cancer biology: AMPK the typical and UBE2O the atypical ????Mol Cell Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28616582 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28616582 #FOAvip English Wikipedia <ref>{{Cite pmid|28616582}}</ref> A new duet in cancer biology: AMPK the typical and UBE2O the atypical #MMPMID28616582 Vila IK; Song SJ; Song MS Mol Cell Oncol 2017[]; 4 (3): ä PMID28616582show ga Ubiquitin-conjugating enzyme E2O (UBE2O) is upregulated in human cancers. We have demonstrated that genetic deletion or pharmacological blockade of UBE2O reduces tumorigenesis through inhibiting the mammalian target of rapamycin complex 1?hypoxia-inducible factor 1-? pathway. Critically, UBE2O targets adenosine monophosphate (AMP)-activated protein kinase-? 2 (AMPK?2) for ubiquitination and degradation. We thus suggest the UBE2O-AMPK?2 axis as a potential therapeutic target for cancer. äA new duet in cancer biology: AMPK the typical and UBE2O the atypical (epmc).pdf DeepDyve Pubget Overpricing