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10.3389/fendo.2017.00119 http://scihub22266oqcxt.onion/10.3389/fendo.2017.00119 C5454061!5454061!28626447     free     free     free       Front+Endocrinol+(Lausanne) 2017 ; 8 (ä): ä Nephropedia Template TP {{tp|p=28626447|t=2017. Autoimmune Thyroiditis and Glomerulopathies |pdf=|usr=}}{{28626447}} gab.com Text Autoimmune Thyroiditis and Glomerulopathies JO: Front Endocrinol (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=28626447 #FOAvip Autoimmune thyroiditis (AIT) is generally associated with hypothyroidism. It affects ~2% of the female population and 0.2% of the male population. The evidence of thyroid function- and thyroid autoantibody-unrelated microproteinuria in almost half of patients with AIT and sometimes heavy proteinuria as in the nephrotic syndrome point to a link of AIT with renal disease. The most common renal diseases observed in AIT are membranous nephropathy, membranoproliferative glomerulonephritis, minimal change disease, IgA nephropathy, focal segmental glomerulosclerosis, antineutrophil cytoplasmic autoantibody (ANCA) vasculitis, and amyloidosis. Different hypotheses have been put forward regarding the relationship between AIT and glomerulopathies, and several potential mechanisms for this association have been considered. Glomerular deposition of immunocomplexes of thyroglobulin and autoantibodies as well as the impaired immune tolerance for megalin (a thyrotropin-regulated glycoprotein expressed on thyroid cells) are the most probable mechanisms. Cross-reactivity between antigens in the setting of genetic predisposition has been considered as a potential mechanism that links the described association between ANCA vasculitis and AIT. Twit Text FOAVip Autoimmune Thyroiditis and Glomerulopathies ????Front Endocrinol (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=28626447 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28626447 #FOAvip English Wikipedia <ref>{{Cite pmid|28626447}}</ref> Autoimmune Thyroiditis and Glomerulopathies #MMPMID28626447 Santoro D; Vadalà C; Siligato R; Buemi M; Benvenga S Front Endocrinol (Lausanne) 2017[]; 8 (ä): ä PMID28626447show ga Autoimmune thyroiditis (AIT) is generally associated with hypothyroidism. It affects ~2% of the female population and 0.2% of the male population. The evidence of thyroid function- and thyroid autoantibody-unrelated microproteinuria in almost half of patients with AIT and sometimes heavy proteinuria as in the nephrotic syndrome point to a link of AIT with renal disease. The most common renal diseases observed in AIT are membranous nephropathy, membranoproliferative glomerulonephritis, minimal change disease, IgA nephropathy, focal segmental glomerulosclerosis, antineutrophil cytoplasmic autoantibody (ANCA) vasculitis, and amyloidosis. Different hypotheses have been put forward regarding the relationship between AIT and glomerulopathies, and several potential mechanisms for this association have been considered. Glomerular deposition of immunocomplexes of thyroglobulin and autoantibodies as well as the impaired immune tolerance for megalin (a thyrotropin-regulated glycoprotein expressed on thyroid cells) are the most probable mechanisms. Cross-reactivity between antigens in the setting of genetic predisposition has been considered as a potential mechanism that links the described association between ANCA vasculitis and AIT. äAutoimmune Thyroiditis and Glomerulopathies (epmc).pdf DeepDyve Pubget Overpricing