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10.1016/j.jcmgh.2017.01.016 http://scihub22266oqcxt.onion/10.1016/j.jcmgh.2017.01.016 C5453879!5453879!28593186     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Cell+Mol+Gastroenterol+Hepatol 2017 ; 4 (1): 153-6 Nephropedia Template TP {{tp|p=28593186|t=2017. Origin of Barrett?s Epithelium: Esophageal Submucosal Glands |pdf=|usr=}}{{28593186}} gab.com Text Origin of Barrett s Epithelium: Esophageal Submucosal Glands JO: Cell Mol Gastroenterol Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=28593186 #FOAvip The origin of the progenitor cell for Barrett?s esophagus remains a major unsolved mystery. Understanding the source of this progenitor may improve strategies to prevent the development of esophageal adenocarcinoma. Esophageal submucosal glands (ESMGs) and ducts may serve as a potential source of progenitor cells that respond to esophageal injury. Through the use of human histologic and molecular analysis, ESMGs and ducts have been described in physical continuity with areas of columnar esophagus, and shared mutations have been described between ESMG ducts and Barrett?s esophagus. Acinar ductal metaplasia, associated with carcinogenesis in other organs, occurs within ESMGs with human esophageal injury and esophageal adenocarcinoma. By using atypical animal models, a squamous epithelial defect well above the gastroesophageal junction healed to columnar epithelium and continuity of ESMG ducts was noted in the new epithelium. Increased proliferation in ESMGs and ducts in response to injury also has been noted in human beings and animals. Twit Text FOAVip Origin of Barrett s Epithelium: Esophageal Submucosal Glands ????Cell Mol Gastroenterol Hepatol http://www.kidney.de/pget.php?&tw=1&pmid=28593186 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28593186 #FOAvip English Wikipedia <ref>{{Cite pmid|28593186}}</ref> Origin of Barrett?s Epithelium: Esophageal Submucosal Glands #MMPMID28593186 Garman KS Cell Mol Gastroenterol Hepatol 2017[Jul]; 4 (1): 153-6 PMID28593186show ga The origin of the progenitor cell for Barrett?s esophagus remains a major unsolved mystery. Understanding the source of this progenitor may improve strategies to prevent the development of esophageal adenocarcinoma. Esophageal submucosal glands (ESMGs) and ducts may serve as a potential source of progenitor cells that respond to esophageal injury. Through the use of human histologic and molecular analysis, ESMGs and ducts have been described in physical continuity with areas of columnar esophagus, and shared mutations have been described between ESMG ducts and Barrett?s esophagus. Acinar ductal metaplasia, associated with carcinogenesis in other organs, occurs within ESMGs with human esophageal injury and esophageal adenocarcinoma. By using atypical animal models, a squamous epithelial defect well above the gastroesophageal junction healed to columnar epithelium and continuity of ESMG ducts was noted in the new epithelium. Increased proliferation in ESMGs and ducts in response to injury also has been noted in human beings and animals. äOrigin of Barrett?s Epithelium: Esophageal Submucosal Glands (epmc).pdf DeepDyve Pubget Overpricing