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10.21037/atm.2017.03.109 http://scihub22266oqcxt.onion/10.21037/atm.2017.03.109 C5451618!5451618!28603721     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Ann+Transl+Med 2017 ; 5 (10): ä Nephropedia Template TP {{tp|p=28603721|t=2017. Helping prometheus: liver protection in acute hemorrhagic shock |pdf=|usr=}}{{28603721}} gab.com Text Helping prometheus: liver protection in acute hemorrhagic shock JO: Ann Transl Med http://www.kidney.de/pget.php?&tw=1&pmid=28603721 #FOAvip Acute hemorrhagic hypovolemic shock is caused by a significant high blood loss and leads to hemodynamic instability. The decrease in intravascular volume results in cellular hypoxia and finally in damage to organs such as the liver and the kidney. The liver plays a decisive role in the development or prevention of multiple organ failure after hemorrhagic shock. Despite the large number of experimental studies, the knowledge of pathophysiological mechanisms in the liver after hemorrhagic shock is incomplete. The aim of this mini review was to provide an overview of the pathophysiological changes in liver function after acute hemorrhagic shock and to address treatment options to improve liver perfusion. Twit Text FOAVip Helping prometheus: liver protection in acute hemorrhagic shock ????Ann Transl Med http://www.kidney.de/pget.php?&tw=1&pmid=28603721 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28603721 #FOAvip English Wikipedia <ref>{{Cite pmid|28603721}}</ref> Helping prometheus: liver protection in acute hemorrhagic shock #MMPMID28603721 Veith NT; Histing T; Menger MD; Pohlemann T; Tschernig T Ann Transl Med 2017[May]; 5 (10): ä PMID28603721show ga Acute hemorrhagic hypovolemic shock is caused by a significant high blood loss and leads to hemodynamic instability. The decrease in intravascular volume results in cellular hypoxia and finally in damage to organs such as the liver and the kidney. The liver plays a decisive role in the development or prevention of multiple organ failure after hemorrhagic shock. Despite the large number of experimental studies, the knowledge of pathophysiological mechanisms in the liver after hemorrhagic shock is incomplete. The aim of this mini review was to provide an overview of the pathophysiological changes in liver function after acute hemorrhagic shock and to address treatment options to improve liver perfusion. äHelping prometheus: liver protection in acute hemorrhagic shock (epmc).pdf DeepDyve Pubget Overpricing