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10.1159/000464422 http://scihub22266oqcxt.onion/10.1159/000464422 C5448443!5448443!28588356     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Ann+Neurosci 2017 ; 24 (1): 46-54 Nephropedia Template TP {{tp|p=28588356|t=2017. Molecular Pathogenesis of Alzheimer s Disease: An Update |pdf=|usr=}}{{28588356}} gab.com Text Molecular Pathogenesis of Alzheimer s Disease: An Update JO: Ann Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=28588356 #FOAvip Dementia is a chronic or progressive syndrome, characterized by impaired cognitive capacity beyond what could be considered a consequence of normal aging. It affects the memory, thinking process, orientation, comprehension, calculation, learning ability, language, and judgment; although awareness is usually unaffected. Alzheimer's disease (AD) is the most common form of dementia; symptoms include memory loss, difficulty solving problems, disorientation in time and space, among others. The disease was first described in 1906 at a conference in Tubingen, Germany by Alois Alzheimer. One hundred and ten years since its first documentation, many aspects of the pathophysiology of AD have been discovered and understood, however gaps of knowledge continue to exist. This literature review summarizes the main underlying neurobiological mechanisms in AD, including the theory with emphasis on amyloid peptide, cholinergic hypothesis, glutamatergic neurotransmission, the role of tau protein, and the involvement of oxidative stress and calcium. Twit Text FOAVip Molecular Pathogenesis of Alzheimer s Disease: An Update ????Ann Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=28588356 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28588356 #FOAvip English Wikipedia <ref>{{Cite pmid|28588356}}</ref> Molecular Pathogenesis of Alzheimer s Disease: An Update #MMPMID28588356 Sanabria-Castro A; Alvarado-Echeverrķa I; Monge-Bonilla C Ann Neurosci 2017[May]; 24 (1): 46-54 PMID28588356show ga Dementia is a chronic or progressive syndrome, characterized by impaired cognitive capacity beyond what could be considered a consequence of normal aging. It affects the memory, thinking process, orientation, comprehension, calculation, learning ability, language, and judgment; although awareness is usually unaffected. Alzheimer's disease (AD) is the most common form of dementia; symptoms include memory loss, difficulty solving problems, disorientation in time and space, among others. The disease was first described in 1906 at a conference in Tubingen, Germany by Alois Alzheimer. One hundred and ten years since its first documentation, many aspects of the pathophysiology of AD have been discovered and understood, however gaps of knowledge continue to exist. This literature review summarizes the main underlying neurobiological mechanisms in AD, including the theory with emphasis on amyloid peptide, cholinergic hypothesis, glutamatergic neurotransmission, the role of tau protein, and the involvement of oxidative stress and calcium. äMolecular Pathogenesis of Alzheimer s Disease: An Update (epmc).pdf DeepDyve Pubget Overpricing