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10.3389/fmed.2017.00064 http://scihub22266oqcxt.onion/10.3389/fmed.2017.00064 C5445129!5445129!28603715     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Med+(Lausanne) 2017 ; 4 (ä): ä Nephropedia Template TP {{tp|p=28603715|t=2017. Contrast-Induced Nephropathy in Renal Transplant Recipients: A Single Center Experience |pdf=|usr=}}{{28603715}} gab.com Text Contrast-Induced Nephropathy in Renal Transplant Recipients: A Single Center Experience JO: Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=28603715 #FOAvip Background: Contrast-induced nephropathy (CIN) in native kidneys is associated with a significant increase in mortality and morbidity. Data regarding CIN in renal allografts are limited, however. We retrospectively studied CIN in renal allografts at our institution: its incidence, risk factors, and effect on long-term outcomes including allograft loss and death. Methods: One hundred thirty-five renal transplant recipients undergoing 161 contrast-enhanced computed tomography (CT) scans or coronary angiograms (Cath) between years 2000 and 2014 were identified. Contrast agents were iso- or low osmolar. CIN was defined as a rise in serum creatinine (SCr) by >0.3?mg/dl or 25% from baseline within 4?days of contrast exposure. After excluding 85 contrast exposures where patients had no SCr within 4?days of contrast administration, 76 exposures (CT: n?=?45; Cath: n?=?31) in 50 eligible patients were analyzed. Risk factors assessed included demographics, comorbid conditions, type/volume of contrast agent used, IV fluids, N-acetylcysteine administration, and calcineurin inhibitor use. Bivariate and multivariable analyses were used to assess the risk of CIN. Results: Incidence of CIN was 13% following both, CT (6 out of 45) and Cath (4 out of 31). Significant bivariate predictors of CIN were IV fluid administration (p?=?0.05), lower hemoglobin (p?=?0.03), and lower albumin (p?=?0.02). In a multivariable model, CIN was predicted by N-acetylcysteine (p?=?0.03) and lower hemoglobin (p?=?0.01). Calcineurin inhibitor use was not associated with CIN. At last follow-up, CIN did not affect allograft or patient survival. Conclusion: CIN is common in kidney transplant recipients, and there is room for quality improvement with regards to careful renal function monitoring post-contrast exposure. In our study, N-acetylcysteine exposure and lower hemoglobin were associated with CIN. Calcineurin inhibitor use was not associated with CIN. Our sample size is small, however, and larger prospective studies of CIN in renal allografts are needed. Twit Text FOAVip Contrast-Induced Nephropathy in Renal Transplant Recipients: A Single Center Experience ????Front Med (Lausanne) http://www.kidney.de/pget.php?&tw=1&pmid=28603715 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28603715 #FOAvip English Wikipedia <ref>{{Cite pmid|28603715}}</ref> Contrast-Induced Nephropathy in Renal Transplant Recipients: A Single Center Experience #MMPMID28603715 Abu Jawdeh BG; Leonard AC; Sharma Y; Katipally S; Shields AR; Alloway RR; Woodle ES; Thakar CV Front Med (Lausanne) 2017[]; 4 (ä): ä PMID28603715show ga Background: Contrast-induced nephropathy (CIN) in native kidneys is associated with a significant increase in mortality and morbidity. Data regarding CIN in renal allografts are limited, however. We retrospectively studied CIN in renal allografts at our institution: its incidence, risk factors, and effect on long-term outcomes including allograft loss and death. Methods: One hundred thirty-five renal transplant recipients undergoing 161 contrast-enhanced computed tomography (CT) scans or coronary angiograms (Cath) between years 2000 and 2014 were identified. Contrast agents were iso- or low osmolar. CIN was defined as a rise in serum creatinine (SCr) by >0.3?mg/dl or 25% from baseline within 4?days of contrast exposure. After excluding 85 contrast exposures where patients had no SCr within 4?days of contrast administration, 76 exposures (CT: n?=?45; Cath: n?=?31) in 50 eligible patients were analyzed. Risk factors assessed included demographics, comorbid conditions, type/volume of contrast agent used, IV fluids, N-acetylcysteine administration, and calcineurin inhibitor use. Bivariate and multivariable analyses were used to assess the risk of CIN. Results: Incidence of CIN was 13% following both, CT (6 out of 45) and Cath (4 out of 31). Significant bivariate predictors of CIN were IV fluid administration (p?=?0.05), lower hemoglobin (p?=?0.03), and lower albumin (p?=?0.02). In a multivariable model, CIN was predicted by N-acetylcysteine (p?=?0.03) and lower hemoglobin (p?=?0.01). Calcineurin inhibitor use was not associated with CIN. At last follow-up, CIN did not affect allograft or patient survival. Conclusion: CIN is common in kidney transplant recipients, and there is room for quality improvement with regards to careful renal function monitoring post-contrast exposure. In our study, N-acetylcysteine exposure and lower hemoglobin were associated with CIN. Calcineurin inhibitor use was not associated with CIN. Our sample size is small, however, and larger prospective studies of CIN in renal allografts are needed. äContrast-Induced Nephropathy in Renal Transplant Recipients: A Single (epmc).pdf DeepDyve Pubget Overpricing