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10.1111/tra.12408 http://scihub22266oqcxt.onion/10.1111/tra.12408 C5444542!5444542!27126894     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Traffic 2016 ; 17 (9): 965-75 Nephropedia Template TP {{tp|p=27126894|t=2016. Herpesvirus entry into host cells mediated by endosomal low pH |pdf=|usr=}}{{27126894}} gab.com Text Herpesvirus entry into host cells mediated by endosomal low pH JO: Traffic http://www.kidney.de/pget.php?&tw=1&pmid=27126894 #FOAvip Herpesviral pathogenesis stems from infection of multiple cell types including the site of latency and cells that support lytic replication. Herpesviruses utilize distinct cellular pathways, including low pH endocytic pathways, to enter different pathophysiologically relevant target cells. This review details the impact of the mildly acidic mileu of endosomes on the entry of herpesviruses, with particular emphasis on herpes simplex virus 1 (HSV-1). Epithelial cells, the portal of primary HSV-1 infection, support entry via low pH endocytosis mechanisms. Mildly acidic pH triggers reversible conformational changes in the HSV-1 class III fusion protein glycoprotein B (gB). In vitro treatment of herpes simplex virions with a similar pH range inactivates infectivity, likely by prematurely activating the viral entry machinery in the absence of a target membrane. How a given herpesvirus mediates both low pH and pH-independent entry events is a key unresolved question. Twit Text FOAVip Herpesvirus entry into host cells mediated by endosomal low pH ????Traffic http://www.kidney.de/pget.php?&tw=1&pmid=27126894 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27126894 #FOAvip English Wikipedia <ref>{{Cite pmid|27126894}}</ref> Herpesvirus entry into host cells mediated by endosomal low pH #MMPMID27126894 Nicola AV Traffic 2016[Sep]; 17 (9): 965-75 PMID27126894show ga Herpesviral pathogenesis stems from infection of multiple cell types including the site of latency and cells that support lytic replication. Herpesviruses utilize distinct cellular pathways, including low pH endocytic pathways, to enter different pathophysiologically relevant target cells. This review details the impact of the mildly acidic mileu of endosomes on the entry of herpesviruses, with particular emphasis on herpes simplex virus 1 (HSV-1). Epithelial cells, the portal of primary HSV-1 infection, support entry via low pH endocytosis mechanisms. Mildly acidic pH triggers reversible conformational changes in the HSV-1 class III fusion protein glycoprotein B (gB). In vitro treatment of herpes simplex virions with a similar pH range inactivates infectivity, likely by prematurely activating the viral entry machinery in the absence of a target membrane. How a given herpesvirus mediates both low pH and pH-independent entry events is a key unresolved question. äHerpesvirus entry into host cells mediated by endosomal low pH (epmc).pdf DeepDyve Pubget Overpricing