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10.5320/wjr.v5.i2.126 http://scihub22266oqcxt.onion/10.5320/wjr.v5.i2.126 C5438095!5438095!28529892     free     free     free Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       World+J+Respirol 2015 ; 5 (2): 126-34 Nephropedia Template TP {{tp|p=28529892|t=2015. Caveolae, caveolin-1 and cavin-1: Emerging roles in pulmonary hypertension |pdf=|usr=}}{{28529892}} gab.com Text Caveolae, caveolin-1 and cavin-1: Emerging roles in pulmonary hypertension JO: World J Respirol http://www.kidney.de/pget.php?&tw=1&pmid=28529892 #FOAvip Caveolae are flask-shaped invaginations of cell membrane that play a significant structural and functional role. Caveolae harbor a variety of signaling molecules and serve to receive, concentrate and transmit extracellular signals across the membrane. Caveolins are the main structural proteins residing in the caveolae. Caveolins and another category of newly identified caveolae regulatory proteins, named cavins, are not only responsible for caveolae formation, but also interact with signaling complexes in the caveolae and regulate transmission of signals across the membrane. In the lung, two of the three caveolin isoforms, i.e., cav-1 and -2, are expressed ubiquitously. Cavin protein family is composed of four proteins, named cavin-1 (or PTRF for polymerase ? and transcript release factor), cavin-2 (or SDPR for serum deprivation protein response), cavin-3 (or SRBC for sdr-related gene product that binds to-c-kinase) and cavin-4 (or MURC for muscle restricted coiled-coiled protein or cavin-4). All the caveolin and cavin proteins are essential regulators for caveolae dynamics. Recently, emerging evidence suggest that caveolae and its associated proteins play crucial roles in development and progression of pulmonary hypertension. The focus of this review is to outline and discuss the contrast in alteration of cav-1 (cav-1),-2 and cavin-1 (PTRF) expression and downstream signaling mechanisms between human and experimental models of pulmonary hypertension. Twit Text FOAVip Caveolae, caveolin-1 and cavin-1: Emerging roles in pulmonary hypertension ????World J Respirol http://www.kidney.de/pget.php?&tw=1&pmid=28529892 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28529892 #FOAvip English Wikipedia <ref>{{Cite pmid|28529892}}</ref> Caveolae, caveolin-1 and cavin-1: Emerging roles in pulmonary hypertension #MMPMID28529892 Chettimada S; Yang J; Moon Hg; Jin Y World J Respirol 2015[Jul]; 5 (2): 126-34 PMID28529892show ga Caveolae are flask-shaped invaginations of cell membrane that play a significant structural and functional role. Caveolae harbor a variety of signaling molecules and serve to receive, concentrate and transmit extracellular signals across the membrane. Caveolins are the main structural proteins residing in the caveolae. Caveolins and another category of newly identified caveolae regulatory proteins, named cavins, are not only responsible for caveolae formation, but also interact with signaling complexes in the caveolae and regulate transmission of signals across the membrane. In the lung, two of the three caveolin isoforms, i.e., cav-1 and -2, are expressed ubiquitously. Cavin protein family is composed of four proteins, named cavin-1 (or PTRF for polymerase ? and transcript release factor), cavin-2 (or SDPR for serum deprivation protein response), cavin-3 (or SRBC for sdr-related gene product that binds to-c-kinase) and cavin-4 (or MURC for muscle restricted coiled-coiled protein or cavin-4). All the caveolin and cavin proteins are essential regulators for caveolae dynamics. Recently, emerging evidence suggest that caveolae and its associated proteins play crucial roles in development and progression of pulmonary hypertension. The focus of this review is to outline and discuss the contrast in alteration of cav-1 (cav-1),-2 and cavin-1 (PTRF) expression and downstream signaling mechanisms between human and experimental models of pulmonary hypertension. äCaveolae, caveolin-1 and cavin-1: Emerging roles in pulmonary hyperten(epmc).pdf DeepDyve Pubget Overpricing