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10.1038/s41598-017-01400-4 http://scihub22266oqcxt.onion/10.1038/s41598-017-01400-4 C5430817!5430817!28446778     free     free     free Deprecated: Implicit conversion from float 225.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 225.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Sci+Rep 2017 ; 7 (ä): ä Nephropedia Template TP {{tp|p=28446778|t=2017. Tumor necrosis factor prevents Candida albicans biofilm formation |pdf=|usr=}}{{28446778}} gab.com Text Tumor necrosis factor prevents Candida albicans biofilm formation JO: Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=28446778 #FOAvip Candida species are commensals but some develop biofilms in prosthetic materials and host surfaces that may represent up to 30% of deaths related to infections, particularly in immunosuppressed patients. Tumor necrosis factor (TNF) exhibits a plethora of functions in host defense mechanisms whereas excessive release of TNF in inflammation promotes tissue damage. Cytokines released in an inflammatory milieu may influence the development of microorganisms either by promoting their growth or displaying antimicrobial activity. In protozoa, TNF may affect growth by coupling through a lectin-like domain, distinct from TNF receptors. TNF was also shown to interact with bacteria via a mechanism that does not involve classical TNF receptors. Using an in vitro C. albicans biofilm model, we show that TNF dose-dependently prevents biofilm development that is blocked by incubating TNF with N,N?-diacetylchitobiose, a major carbohydrate component of C. albicans cell wall. This finding represents a relevant and hitherto unknown mechanism that adds to the understanding of why TNF blockade is associated with opportunistic C. albicans infections. Twit Text FOAVip Tumor necrosis factor prevents Candida albicans biofilm formation ????Sci Rep http://www.kidney.de/pget.php?&tw=1&pmid=28446778 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28446778 #FOAvip English Wikipedia <ref>{{Cite pmid|28446778}}</ref> Tumor necrosis factor prevents Candida albicans biofilm formation #MMPMID28446778 Rocha FAC; Alves AMCV; Rocha MFG; Cordeiro Rde A; Brilhante RSN; Pinto ACMD; Nunes Rde M; Girão VCC; Sidrim JJC Sci Rep 2017[]; 7 (ä): ä PMID28446778show ga Candida species are commensals but some develop biofilms in prosthetic materials and host surfaces that may represent up to 30% of deaths related to infections, particularly in immunosuppressed patients. Tumor necrosis factor (TNF) exhibits a plethora of functions in host defense mechanisms whereas excessive release of TNF in inflammation promotes tissue damage. Cytokines released in an inflammatory milieu may influence the development of microorganisms either by promoting their growth or displaying antimicrobial activity. In protozoa, TNF may affect growth by coupling through a lectin-like domain, distinct from TNF receptors. TNF was also shown to interact with bacteria via a mechanism that does not involve classical TNF receptors. Using an in vitro C. albicans biofilm model, we show that TNF dose-dependently prevents biofilm development that is blocked by incubating TNF with N,N?-diacetylchitobiose, a major carbohydrate component of C. albicans cell wall. This finding represents a relevant and hitherto unknown mechanism that adds to the understanding of why TNF blockade is associated with opportunistic C. albicans infections. äTumor necrosis factor prevents Candida albicans biofilm formation (epmc).pdf DeepDyve Pubget Overpricing