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10.1093/gastro/gox013 http://scihub22266oqcxt.onion/10.1093/gastro/gox013 C5421503!5421503!28533911     free     free     free       Gastroenterol+Rep+(Oxf) 2017 ; 5 (2): 138-47 Nephropedia Template TP {{tp|p=28533911|t=2017. Hepatic encephalopathy |pdf=|usr=}}{{28533911}} gab.com Text Hepatic encephalopathy JO: Gastroenterol Rep (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=28533911 #FOAvip Hepatic encephalopathy (HE) is a reversible syndrome of impaired brain function occurring in patients with advanced liver diseases. The precise pathophysiology of HE is still under discussion; the leading hypothesis focus on the role of neurotoxins, impaired neurotransmission due to metabolic changes in liver failure, changes in brain energy metabolism, systemic inflammatory response and alterations of the blood brain barrier. HE produces a wide spectrum of nonspecific neurological and psychiatric manifestations. Minimal HE is diagnosed by abnormal psychometric tests. Clinically overt HE includes personality changes, alterations in consciousness progressive disorientation in time and space, somnolence, stupor and, finally, coma. Except for clinical studies, no specific tests are required for diagnosis. HE is classified according to the underlying disease, the severity of manifestations, its time course and the existence of precipitating factors. Treatment of overt HE includes supportive therapies, treatment of precipitating factors, lactulose and/or rifaximin. Routine treatment for minimal HE is only recommended for selected patients. Twit Text FOAVip Hepatic encephalopathy ????Gastroenterol Rep (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=28533911 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28533911 #FOAvip English Wikipedia <ref>{{Cite pmid|28533911}}</ref> Hepatic encephalopathy #MMPMID28533911 Ferenci P Gastroenterol Rep (Oxf) 2017[May]; 5 (2): 138-47 PMID28533911show ga Hepatic encephalopathy (HE) is a reversible syndrome of impaired brain function occurring in patients with advanced liver diseases. The precise pathophysiology of HE is still under discussion; the leading hypothesis focus on the role of neurotoxins, impaired neurotransmission due to metabolic changes in liver failure, changes in brain energy metabolism, systemic inflammatory response and alterations of the blood brain barrier. HE produces a wide spectrum of nonspecific neurological and psychiatric manifestations. Minimal HE is diagnosed by abnormal psychometric tests. Clinically overt HE includes personality changes, alterations in consciousness progressive disorientation in time and space, somnolence, stupor and, finally, coma. Except for clinical studies, no specific tests are required for diagnosis. HE is classified according to the underlying disease, the severity of manifestations, its time course and the existence of precipitating factors. Treatment of overt HE includes supportive therapies, treatment of precipitating factors, lactulose and/or rifaximin. Routine treatment for minimal HE is only recommended for selected patients. äHepatic encephalopathy (epmc).pdf DeepDyve Pubget Overpricing