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10.1093/gastro/gox009 http://scihub22266oqcxt.onion/10.1093/gastro/gox009 C5421450!5421450!28533910     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Gastroenterol+Rep+(Oxf) 2017 ; 5 (2): 127-37 Nephropedia Template TP {{tp|p=28533910|t=2017. Renal dysfunction in cirrhosis: acute kidney injury and the hepatorenal syndrome |pdf=|usr=}}{{28533910}} gab.com Text Renal dysfunction in cirrhosis: acute kidney injury and the hepatorenal syndrome JO: Gastroenterol Rep (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=28533910 #FOAvip Renal dysfunction is a common complication of liver cirrhosis and of utmost clinical and prognostic relevance. Patients with cirrhosis are more prone to developing acute kidney injury (AKI) than the non-cirrhotic population. Pre-renal AKI, the hepatorenal syndrome type of AKI (HRS-AKI, formerly known as ?type 1?) and acute tubular necrosis represent the most common causes of AKI in cirrhosis. Correct differentiation is imperative, as treatment differs substantially. While pre-renal AKI usually responds well to plasma volume expansion, HRS-AKI and ATN require different specific approaches and are associated with substantial mortality. Several paradigms, such as the threshold of 2.5?mg/dL for diagnosis of HRS-AKI, have recently been abolished and novel urinary biomarkers are being investigated in order to facilitate early and correct diagnosis and treatment of HRS-AKI and other forms of AKI in patients with cirrhosis. This review summarizes the current diagnostic criteria, as well as pathophysiologic and therapeutic concepts for AKI and HRS-AKI in cirrhosis. Twit Text FOAVip Renal dysfunction in cirrhosis: acute kidney injury and the hepatorenal syndrome ????Gastroenterol Rep (Oxf) http://www.kidney.de/pget.php?&tw=1&pmid=28533910 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28533910 #FOAvip English Wikipedia <ref>{{Cite pmid|28533910}}</ref> Renal dysfunction in cirrhosis: acute kidney injury and the hepatorenal syndrome #MMPMID28533910 Bucsics T; Krones E Gastroenterol Rep (Oxf) 2017[May]; 5 (2): 127-37 PMID28533910show ga Renal dysfunction is a common complication of liver cirrhosis and of utmost clinical and prognostic relevance. Patients with cirrhosis are more prone to developing acute kidney injury (AKI) than the non-cirrhotic population. Pre-renal AKI, the hepatorenal syndrome type of AKI (HRS-AKI, formerly known as ?type 1?) and acute tubular necrosis represent the most common causes of AKI in cirrhosis. Correct differentiation is imperative, as treatment differs substantially. While pre-renal AKI usually responds well to plasma volume expansion, HRS-AKI and ATN require different specific approaches and are associated with substantial mortality. Several paradigms, such as the threshold of 2.5?mg/dL for diagnosis of HRS-AKI, have recently been abolished and novel urinary biomarkers are being investigated in order to facilitate early and correct diagnosis and treatment of HRS-AKI and other forms of AKI in patients with cirrhosis. This review summarizes the current diagnostic criteria, as well as pathophysiologic and therapeutic concepts for AKI and HRS-AKI in cirrhosis. äRenal dysfunction in cirrhosis: acute kidney injury and the hepatorena(epmc).pdf DeepDyve Pubget Overpricing