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10.1155/2017/3560238 http://scihub22266oqcxt.onion/10.1155/2017/3560238 C5420432!5420432 !28512641     free     free     free Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28512641 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Diabetes+Res 2017 ; 2017 (ä): 3560238 Nephropedia Template TP {{tp|p=28512641 |t=2017. Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy |pdf=|usr=}}{{28512641 }} gab.com Text Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy JO: J Diabetes Res http://www.kidney.de/pget.php?&tw=1&pmid=28512641 #FOAvip Diabetic nephropathy (DN), a leading cause of end-stage renal disease (ESRD), becomes a worldwide problem. Ultrastructural changes of the glomerular filtration barrier, especially the pathological changes of podocytes, lead to proteinuria in patients with diabetes. Podocytes are major components of glomerular filtration barrier, lining outside of the glomerular basement membrane (GBM) to maintain the permeability of the GBM. Autophagy is a high conserved cellular process in lysosomes including impaired protein, cell organelles, and other contents in the cytoplasm. Recent studies suggest that activation of autophagy in podocytes may be a potential therapy to prevent the progression of DN. Here, we review the mechanisms of autophagy in podocytes and discuss the current studies about alleviating proteinuria via activating podocyte autophagy. Twit Text FOAVip Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy ????J Diabetes Res http://www.kidney.de/pget.php?&tw=1&pmid=28512641 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28512641 #FOAvip English Wikipedia <ref>{{Cite pmid|28512641 }}</ref> Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Progression of Diabetic Nephropathy #MMPMID28512641 Liu N ; Xu L ; Shi Y ; Zhuang S J Diabetes Res 2017[]; 2017 (ä): 3560238 PMID28512641 show ga Diabetic nephropathy (DN), a leading cause of end-stage renal disease (ESRD), becomes a worldwide problem. Ultrastructural changes of the glomerular filtration barrier, especially the pathological changes of podocytes, lead to proteinuria in patients with diabetes. Podocytes are major components of glomerular filtration barrier, lining outside of the glomerular basement membrane (GBM) to maintain the permeability of the GBM. Autophagy is a high conserved cellular process in lysosomes including impaired protein, cell organelles, and other contents in the cytoplasm. Recent studies suggest that activation of autophagy in podocytes may be a potential therapy to prevent the progression of DN. Here, we review the mechanisms of autophagy in podocytes and discuss the current studies about alleviating proteinuria via activating podocyte autophagy. |*Autophagy [MESH] |*Podocytes [MESH] |AMP-Activated Protein Kinases/metabolism [MESH] |Class I Phosphatidylinositol 3-Kinases/metabolism [MESH] |Diabetic Nephropathies/*metabolism [MESH] |Disease Progression [MESH] |Humans [MESH] |Kidney Failure, Chronic/metabolism [MESH] |Proteinuria/*metabolism [MESH] |Proto-Oncogene Proteins c-akt/metabolism [MESH] |Reactive Oxygen Species/metabolism [MESH] |Renal Insufficiency, Chronic/*metabolism [MESH] |Signal Transduction [MESH]Podocyte Autophagy: A Potential Therapeutic Target to Prevent the Prog(epmc).pdf DeepDyve Pubget Overpricing