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10.1007/s00232-016-9881-6 http://scihub22266oqcxt.onion/10.1007/s00232-016-9881-6 C5417354!5417354!26914707     free     free     free Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 233.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Membr+Biol 2016 ; 249 (3): 199-213 Nephropedia Template TP {{tp|p=26914707|t=2016. Connexins, pannexins and their channels in fibroproliferative diseases |pdf=|usr=}}{{26914707}} gab.com Text Connexins, pannexins and their channels in fibroproliferative diseases JO: J Membr Biol http://www.kidney.de/pget.php?&tw=1&pmid=26914707 #FOAvip Cellular and molecular mechanisms of wound healing, tissue repair and fibrogenesis are established in different organs and are essential for the maintenance of function and tissue integrity after cell injury. These mechanisms are also involved in a plethora of fibroproliferative diseases or organ-specific fibrotic disorders, all of which are associated with the excessive deposition of extracellular matrix components. Fibroblasts, which are key cells in tissue repair and fibrogenesis, rely on communicative cellular networks to ensure efficient control of these processes and to prevent abnormal accumulation of extracellular matrix into the tissue. Despite the significant impact on human health, and thus the epidemiologic relevance, there is still no effective treatment for most fibrosis-related diseases. This paper provides an overview of current concepts and mechanisms involved in the participation of cellular communication via connexin-based pores as well as pannexin-based channels in the processes of tissue repair and fibrogenesis in chronic diseases. Understanding these mechanisms may contribute to the development of new therapeutic strategies to clinically manage fibroproliferative diseases and organ-specific fibrotic disorders. Twit Text FOAVip Connexins, pannexins and their channels in fibroproliferative diseases ????J Membr Biol http://www.kidney.de/pget.php?&tw=1&pmid=26914707 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26914707 #FOAvip English Wikipedia <ref>{{Cite pmid|26914707}}</ref> Connexins, pannexins and their channels in fibroproliferative diseases #MMPMID26914707 Cogliati B; Mennecier G; Willebrords J; Da Silva TC; Maes M; Pereira IVA; Crespo-Yanguas S; Hernandez-Blazquez FJ; Dagli MLZ; Vinken M J Membr Biol 2016[Jun]; 249 (3): 199-213 PMID26914707show ga Cellular and molecular mechanisms of wound healing, tissue repair and fibrogenesis are established in different organs and are essential for the maintenance of function and tissue integrity after cell injury. These mechanisms are also involved in a plethora of fibroproliferative diseases or organ-specific fibrotic disorders, all of which are associated with the excessive deposition of extracellular matrix components. Fibroblasts, which are key cells in tissue repair and fibrogenesis, rely on communicative cellular networks to ensure efficient control of these processes and to prevent abnormal accumulation of extracellular matrix into the tissue. Despite the significant impact on human health, and thus the epidemiologic relevance, there is still no effective treatment for most fibrosis-related diseases. This paper provides an overview of current concepts and mechanisms involved in the participation of cellular communication via connexin-based pores as well as pannexin-based channels in the processes of tissue repair and fibrogenesis in chronic diseases. Understanding these mechanisms may contribute to the development of new therapeutic strategies to clinically manage fibroproliferative diseases and organ-specific fibrotic disorders. äConnexins, pannexins and their channels in fibroproliferative diseases(epmc).pdf DeepDyve Pubget Overpricing