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10.1177/1559325817699697 http://scihub22266oqcxt.onion/10.1177/1559325817699697 C5407528!5407528!28473742     free     free     free Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Dose+Response 2017 ; 15 (1): ä Nephropedia Template TP {{tp|p=28473742|t=2017. NADPH Oxidase Activation Contributes to Heavy Ion Irradiation?Induced Cell Death |pdf=|usr=}}{{28473742}} gab.com Text NADPH Oxidase Activation Contributes to Heavy Ion Irradiation Induced Cell Death JO: Dose Response http://www.kidney.de/pget.php?&tw=1&pmid=28473742 #FOAvip Increased oxidative stress plays an important role in heavy ion radiation?induced cell death. The mechanism involved in the generation of elevated reactive oxygen species (ROS) is not fully illustrated. Here we show that NADPH oxidase activation is closely related to heavy ion radiation?induced cell death via excessive ROS generation. Cell death and cellular ROS can be greatly reduced in irradiated cancer cells with the preincubation of diphenyleneiodium, an inhibitor of NADPH oxidase. Most of the NADPH oxidase (NOX) family proteins (NOX1, NOX2, NOX3, NOX4, and NOX5) showed increased expression after heavy ion irradiation. Meanwhile, the cytoplasmic subunit p47phox was translocated to the cell membrane and localized with NOX2 to form reactive NADPH oxidase. Our data suggest for the first time that ROS generation, as mediated by NADPH oxidase activation, could be an important contributor to heavy ion irradiation?induced cell death. Twit Text FOAVip NADPH Oxidase Activation Contributes to Heavy Ion Irradiation Induced Cell Death ????Dose Response http://www.kidney.de/pget.php?&tw=1&pmid=28473742 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28473742 #FOAvip English Wikipedia <ref>{{Cite pmid|28473742}}</ref> NADPH Oxidase Activation Contributes to Heavy Ion Irradiation?Induced Cell Death #MMPMID28473742 Wang Y; Liu Q; Zhao W; Zhou X; Miao G; Sun C; Zhang H Dose Response 2017[Jan]; 15 (1): ä PMID28473742show ga Increased oxidative stress plays an important role in heavy ion radiation?induced cell death. The mechanism involved in the generation of elevated reactive oxygen species (ROS) is not fully illustrated. Here we show that NADPH oxidase activation is closely related to heavy ion radiation?induced cell death via excessive ROS generation. Cell death and cellular ROS can be greatly reduced in irradiated cancer cells with the preincubation of diphenyleneiodium, an inhibitor of NADPH oxidase. Most of the NADPH oxidase (NOX) family proteins (NOX1, NOX2, NOX3, NOX4, and NOX5) showed increased expression after heavy ion irradiation. Meanwhile, the cytoplasmic subunit p47phox was translocated to the cell membrane and localized with NOX2 to form reactive NADPH oxidase. Our data suggest for the first time that ROS generation, as mediated by NADPH oxidase activation, could be an important contributor to heavy ion irradiation?induced cell death. äNADPH Oxidase Activation Contributes to Heavy Ion Irradiation?Induced (epmc).pdf DeepDyve Pubget Overpricing