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10.3390/jcm6040041 http://scihub22266oqcxt.onion/10.3390/jcm6040041 C5406773!5406773!28368299     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       J+Clin+Med 2017 ; 6 (4): ä Nephropedia Template TP {{tp|p=28368299|t=2017. Natural Killer T Cells in Liver Ischemia?Reperfusion Injury |pdf=|usr=}}{{28368299}} gab.com Text Natural Killer T Cells in Liver Ischemia Reperfusion Injury JO: J Clin Med http://www.kidney.de/pget.php?&tw=1&pmid=28368299 #FOAvip Restoration of blood flow to an ischemic organ results in significant tissue injury. In the field of liver transplantation, ischemia?reperfusion injury (IRI) has proven to be a formidable clinical obstacle. In addition to metabolic stress and inflammation, IRI results in profound graft dysfunction and loss. The severity of IRI further limits the ability to expand the donor pool by using partial grafts and marginal organs. As such, the inflammatory response to reperfusion of the liver continues to be an area of intense investigation. Among the various leukocytes involved in IRI, new insights suggest that natural killer T (NKT) cells may be a central driver of hepatocellular injury. Herein, we examine recent experimental observations that provide a mechanistic link between NKT cell recruitment to liver and post-perfusion tissue injury. Twit Text FOAVip Natural Killer T Cells in Liver Ischemia Reperfusion Injury ????J Clin Med http://www.kidney.de/pget.php?&tw=1&pmid=28368299 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28368299 #FOAvip English Wikipedia <ref>{{Cite pmid|28368299}}</ref> Natural Killer T Cells in Liver Ischemia?Reperfusion Injury #MMPMID28368299 Zimmerman MA; Martin A; Yee J; Schiller J; Hong JC J Clin Med 2017[Apr]; 6 (4): ä PMID28368299show ga Restoration of blood flow to an ischemic organ results in significant tissue injury. In the field of liver transplantation, ischemia?reperfusion injury (IRI) has proven to be a formidable clinical obstacle. In addition to metabolic stress and inflammation, IRI results in profound graft dysfunction and loss. The severity of IRI further limits the ability to expand the donor pool by using partial grafts and marginal organs. As such, the inflammatory response to reperfusion of the liver continues to be an area of intense investigation. Among the various leukocytes involved in IRI, new insights suggest that natural killer T (NKT) cells may be a central driver of hepatocellular injury. Herein, we examine recent experimental observations that provide a mechanistic link between NKT cell recruitment to liver and post-perfusion tissue injury. äNatural Killer T Cells in Liver Ischemia?Reperfusion Injury (epmc).pdf DeepDyve Pubget Overpricing