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10.1016/bs.ctm.2016.05.001 http://scihub22266oqcxt.onion/10.1016/bs.ctm.2016.05.001 C5398315!5398315!27586288     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27586288&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Curr+Top+Membr 2016 ; 78 (ä): 287-311 Nephropedia Template TP {{tp|p=27586288|t=2016. Cardiac Na Channels: Structure to Function |pdf=|usr=}}{{27586288}} gab.com Text Cardiac Na Channels: Structure to Function JO: Curr Top Membr http://www.kidney.de/pget.php?&tw=1&pmid=27586288 #FOAvip Heart rhythms arise from electrical activity generated by precisely timed opening and closing of ion channels in individual cardiac myocytes. Opening of the primary cardiac voltage-gated sodium (Nav1.5) channel initiates cellular depolarization and the propagation of an electrical action potential that promotes coordinated contraction of the heart. The regularity of these contractile waves is critically important since it drives the primary function of the heart: to act as a pump that delivers blood to the brain and vital organs. When electrical activity goes awry during a cardiac arrhythmia, the pump does not function, the brain does not receive oxygenated blood, and death ensues. Perturbations to NaV1.5 may alter the structure, and hence the function, of the ion channel and are associated downstream with a wide variety of cardiac conduction pathologies, such as arrhythmias. Twit Text FOAVip Cardiac Na Channels: Structure to Function ????Curr Top Membr http://www.kidney.de/pget.php?&tw=1&pmid=27586288 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27586288 #FOAvip English Wikipedia <ref>{{Cite pmid|27586288}}</ref> Cardiac Na Channels: Structure to Function #MMPMID27586288 DeMarco K; Clancy C Curr Top Membr 2016[]; 78 (ä): 287-311 PMID27586288show ga Heart rhythms arise from electrical activity generated by precisely timed opening and closing of ion channels in individual cardiac myocytes. Opening of the primary cardiac voltage-gated sodium (Nav1.5) channel initiates cellular depolarization and the propagation of an electrical action potential that promotes coordinated contraction of the heart. The regularity of these contractile waves is critically important since it drives the primary function of the heart: to act as a pump that delivers blood to the brain and vital organs. When electrical activity goes awry during a cardiac arrhythmia, the pump does not function, the brain does not receive oxygenated blood, and death ensues. Perturbations to NaV1.5 may alter the structure, and hence the function, of the ion channel and are associated downstream with a wide variety of cardiac conduction pathologies, such as arrhythmias. äCardiac Na Channels: Structure to Function (epmc).pdf DeepDyve Pubget Overpricing