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10.3389/fneur.2017.00141 http://scihub22266oqcxt.onion/10.3389/fneur.2017.00141 C5397512!5397512!28473800     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Front+Neurol 2017 ; 8 (ä): ä Nephropedia Template TP {{tp|p=28473800|t=2017. Central Positional Nystagmus: A Systematic Literature Review |pdf=|usr=}}{{28473800}} gab.com Text Central Positional Nystagmus: A Systematic Literature Review JO: Front Neurol http://www.kidney.de/pget.php?&tw=1&pmid=28473800 #FOAvip Objective: To provide a systematic review of the clinical and radiological features of lesion-induced central positional nystagmus (CPN) and identify salient characteristics that differentiate central from peripheral positional nystagmus (PN). Methods: Systematic literature search according to the preferred reporting items for systematic reviews and meta-analysis. Results: A total of 82 patients from 28 studies met the participants intervention, comparison, outcomes, and study designs criteria for inclusion. An atypical direction of nystagmus for the stimulated canal was reported in 97.5% patients during Dix?Hallpike (D?H) and 54.5% upon supine roll testing. Five types of CPNs were identified during positional testing: positional horizontal nystagmus (pHN) (36.8%), positional downbeating nystagmus (pDBN) (29.2%), positional torsional nystagmus (pTN) (2.1%), positional upbeating nystagmus (pUBN) (2.1%), and a combination of the four profiles (29.9%). CPN was paroxysmal (<60?s) in 85% patients on straight head hanging (SHH), 63.9% on D?H, and 37.5% on supine roll, and had a latency <3?s upon positioning in 94.7% patients in which it was reported. Concurrent vertigo was reportedly present in 63.4% patients and 48.8% demonstrated other neurological signs. Radiologically, in 74.4%, there was mention of cerebellar involvement, isolated brainstem involvement in 8.5%, and 14.6% involved the fourth ventricle. Conclusion: Currently, there is a lack of robust data on the clinical and radiological characteristics of CPN highlighting the need for better phenotyping of CPN to help differentiate this entity from peripheral causes of PN. With increased awareness of CPN, particularly in the acute setting, we may see a change in the estimated prevalence of CPN and improved clinical markers to promptly identify the frequently sinister underlying causes. Twit Text FOAVip Central Positional Nystagmus: A Systematic Literature Review ????Front Neurol http://www.kidney.de/pget.php?&tw=1&pmid=28473800 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28473800 #FOAvip English Wikipedia <ref>{{Cite pmid|28473800}}</ref> Central Positional Nystagmus: A Systematic Literature Review #MMPMID28473800 Macdonald NK; Kaski D; Saman Y; Al-Shaikh Sulaiman A; Anwer A; Bamiou DE Front Neurol 2017[]; 8 (ä): ä PMID28473800show ga Objective: To provide a systematic review of the clinical and radiological features of lesion-induced central positional nystagmus (CPN) and identify salient characteristics that differentiate central from peripheral positional nystagmus (PN). Methods: Systematic literature search according to the preferred reporting items for systematic reviews and meta-analysis. Results: A total of 82 patients from 28 studies met the participants intervention, comparison, outcomes, and study designs criteria for inclusion. An atypical direction of nystagmus for the stimulated canal was reported in 97.5% patients during Dix?Hallpike (D?H) and 54.5% upon supine roll testing. Five types of CPNs were identified during positional testing: positional horizontal nystagmus (pHN) (36.8%), positional downbeating nystagmus (pDBN) (29.2%), positional torsional nystagmus (pTN) (2.1%), positional upbeating nystagmus (pUBN) (2.1%), and a combination of the four profiles (29.9%). CPN was paroxysmal (<60?s) in 85% patients on straight head hanging (SHH), 63.9% on D?H, and 37.5% on supine roll, and had a latency <3?s upon positioning in 94.7% patients in which it was reported. Concurrent vertigo was reportedly present in 63.4% patients and 48.8% demonstrated other neurological signs. Radiologically, in 74.4%, there was mention of cerebellar involvement, isolated brainstem involvement in 8.5%, and 14.6% involved the fourth ventricle. Conclusion: Currently, there is a lack of robust data on the clinical and radiological characteristics of CPN highlighting the need for better phenotyping of CPN to help differentiate this entity from peripheral causes of PN. With increased awareness of CPN, particularly in the acute setting, we may see a change in the estimated prevalence of CPN and improved clinical markers to promptly identify the frequently sinister underlying causes. äCentral Positional Nystagmus: A Systematic Literature Review (epmc).pdf DeepDyve Pubget Overpricing