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10.1155/2017/8415179 http://scihub22266oqcxt.onion/10.1155/2017/8415179 C5385902!5385902 !28523279     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28523279 &cmd=llinks): Failed to open stream: HTTP request failed! 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Increased Neutrophil Extracellular Trap Formation in Uremia Is Associated with Chronic Inflammation and Prevalent Coronary Artery Disease |pdf=|usr=}}{{28523279 }} gab.com Text Increased Neutrophil Extracellular Trap Formation in Uremia Is Associated with Chronic Inflammation and Prevalent Coronary Artery Disease JO: J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=28523279 #FOAvip Background. Neutrophils are involved in the pathogenesis of atherosclerosis by neutrophil extracellular trap (NET) formation. We hypothesized that the NET formation of neutrophils might be changed in end-stage renal disease (ESRD) patients, explaining their higher incidence of coronary artery diseases (CAD). Method. A cross-sectional study was performed in 60 maintenance hemodialysis (MHD) patients, 30 age- and sex-matched healthy individuals (HV, negative control), and 30 patients with acute infection (positive control). Neutrophil activation and function were measured with reactive oxygen species (ROS) activity, degranulation, NET formation, and phenotypical changes. Result. Compared with HV, neutrophils extracted from MHD patients displayed significantly increased levels of basal NET formation, ROS production, and degranulation, suggesting spontaneous activation in uremia. Also, an increase in citrullinated histone H3 was detected in this group compared to the HV. And neutrophils from HV were normal CD16(bright)/CD62L(bright) cells; however, neutrophils from MHD were CD16(bright)/CD62L(dim), similar to those from patients with acute infections. Interestingly, multivariate analyses identified the prevalent CAD and neutrophil counts as independent determinants of baseline NET formation (? = 0.323, p = 0.016 and ? = 0.369, p = 0.006, resp.). Conclusions. Uremia-associated-increased NET formation may be a sign of increased burden of atherosclerosis. Twit Text FOAVip Increased Neutrophil Extracellular Trap Formation in Uremia Is Associated with Chronic Inflammation and Prevalent Coronary Artery Disease ????J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=28523279 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28523279 #FOAvip English Wikipedia <ref>{{Cite pmid|28523279 }}</ref> Increased Neutrophil Extracellular Trap Formation in Uremia Is Associated with Chronic Inflammation and Prevalent Coronary Artery Disease #MMPMID28523279 Kim JK ; Hong CW ; Park MJ ; Song YR ; Kim HJ ; Kim SG J Immunol Res 2017[]; 2017 (ä): 8415179 PMID28523279 show ga Background. Neutrophils are involved in the pathogenesis of atherosclerosis by neutrophil extracellular trap (NET) formation. We hypothesized that the NET formation of neutrophils might be changed in end-stage renal disease (ESRD) patients, explaining their higher incidence of coronary artery diseases (CAD). Method. A cross-sectional study was performed in 60 maintenance hemodialysis (MHD) patients, 30 age- and sex-matched healthy individuals (HV, negative control), and 30 patients with acute infection (positive control). Neutrophil activation and function were measured with reactive oxygen species (ROS) activity, degranulation, NET formation, and phenotypical changes. Result. Compared with HV, neutrophils extracted from MHD patients displayed significantly increased levels of basal NET formation, ROS production, and degranulation, suggesting spontaneous activation in uremia. Also, an increase in citrullinated histone H3 was detected in this group compared to the HV. And neutrophils from HV were normal CD16(bright)/CD62L(bright) cells; however, neutrophils from MHD were CD16(bright)/CD62L(dim), similar to those from patients with acute infections. Interestingly, multivariate analyses identified the prevalent CAD and neutrophil counts as independent determinants of baseline NET formation (? = 0.323, p = 0.016 and ? = 0.369, p = 0.006, resp.). Conclusions. Uremia-associated-increased NET formation may be a sign of increased burden of atherosclerosis. |*Extracellular Traps [MESH] |Coronary Artery Disease/*etiology/immunology/physiopathology [MESH] |Cross-Sectional Studies [MESH] |Female [MESH] |Histones/blood [MESH] |Humans [MESH] |Inflammation/*pathology [MESH] |Kidney Failure, Chronic/complications/immunology/pathology [MESH] |Male [MESH] |Neutrophil Activation [MESH] |Neutrophils/drug effects/*immunology [MESH] |Prevalence [MESH] |Reactive Oxygen Species/analysis [MESH] |Tetradecanoylphorbol Acetate/pharmacology [MESH]Increased Neutrophil Extracellular Trap Formation in Uremia Is Associa(epmc).pdf DeepDyve Pubget Overpricing