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10.1007/s00281-013-0415-3 http://scihub22266oqcxt.onion/10.1007/s00281-013-0415-3 C5385209!5385209!24402710     free     free     free Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 229.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534       Semin+Immunopathol 2014 ; 36 (4): 421-9 Nephropedia Template TP {{tp|p=24402710|t=2014. Immunopathogenesis of idiopathic nephrotic syndrome with relapse |pdf=|usr=}}{{24402710}} gab.com Text Immunopathogenesis of idiopathic nephrotic syndrome with relapse JO: Semin Immunopathol http://www.kidney.de/pget.php?&tw=1&pmid=24402710 #FOAvip Idiopathic change nephrotic syndrome (INS), the most frequent glomerular disease in children and young adults, is characterized by heavy proteinuria and a relapsing remitting course. Although the mechanisms underlying the pathophysiology of proteinuria remain unclear, clinical and experimental observations suggest that lymphocyte and podocyte disturbances are two sides of the disease. The current hypothesis suggests that immune cells release a putative factor, which alters podocyte function resulting in nephrotic proteinuria. Besides T cell abnormalities, recent evidence of B cell depletion efficacy in sustained remissions added a new challenge in understanding the immunological mechanisms of INS. In this review, we discuss recent insights related to podocyte disorders occurring in INS and their relevance in human diseases. Twit Text FOAVip Immunopathogenesis of idiopathic nephrotic syndrome with relapse ????Semin Immunopathol http://www.kidney.de/pget.php?&tw=1&pmid=24402710 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24402710 #FOAvip English Wikipedia <ref>{{Cite pmid|24402710}}</ref> Immunopathogenesis of idiopathic nephrotic syndrome with relapse #MMPMID24402710 Sahali D; Sendeyo K; Mangier M; Audard V; Zhang SY; Lang P; Ollero M; Pawlak A Semin Immunopathol 2014[Jul]; 36 (4): 421-9 PMID24402710show ga Idiopathic change nephrotic syndrome (INS), the most frequent glomerular disease in children and young adults, is characterized by heavy proteinuria and a relapsing remitting course. Although the mechanisms underlying the pathophysiology of proteinuria remain unclear, clinical and experimental observations suggest that lymphocyte and podocyte disturbances are two sides of the disease. The current hypothesis suggests that immune cells release a putative factor, which alters podocyte function resulting in nephrotic proteinuria. Besides T cell abnormalities, recent evidence of B cell depletion efficacy in sustained remissions added a new challenge in understanding the immunological mechanisms of INS. In this review, we discuss recent insights related to podocyte disorders occurring in INS and their relevance in human diseases. äImmunopathogenesis of idiopathic nephrotic syndrome with relapse (epmc).pdf DeepDyve Pubget Overpricing